推荐产品
化驗
98%
形狀
liquid
折射率
n20/D 1.534 (lit.)
bp
220 °C (lit.)
mp
11-13 °C (lit.)
密度
1.102 g/mL at 25 °C (lit.)
SMILES 字串
CC(=O)c1cccnc1
InChI
1S/C7H7NO/c1-6(9)7-3-2-4-8-5-7/h2-5H,1H3
InChI 密鑰
WEGYGNROSJDEIW-UHFFFAOYSA-N
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訊號詞
Danger
危險聲明
危險分類
Acute Tox. 3 Oral - Skin Irrit. 2
儲存類別代碼
6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects
水污染物質分類（WGK）
WGK 1
閃點（°F）
219.2 °F - closed cup
閃點（°C）
104 °C - closed cup
個人防護裝備
Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter
其他客户在看
Matasha Dhar et al.
Nature communications, 7, 12938-12938 (2016-09-22)
Neurodegenerative lesions induce sprouting of new collaterals from surviving axons, but the extent to which this form of axonal remodelling alters brain functional structure remains unclear. To understand how collateral sprouting proceeds in the adult brain, we imaged post-lesion sprouting
Iran Goudarzi et al.
European journal of pharmacology, 642(1-3), 56-65 (2010-07-20)
Electrophysiological dysfunction of Purkinje cells causes cerebellar ataxia. Recent studies indicated that 4-aminopyridine (4-AP) can prevent the attacks in patients with episodic ataxia type 2. However, the cellular mechanism(s) by which 4-AP might be beneficial for the improvement of motor
Yi Zhang et al.
American journal of physiology. Heart and circulatory physiology, 285(3), H1177-H1182 (2003-05-17)
We sought to define the contribution of the climbing fibers (CF), one of the major inputs to Purkinje neurons, to the increase in cerebellar blood flow (BFcrb) produced by activation of the cerebellar cortex. The neurotoxin 3-acetylpyridine was used to
Brian H Hess et al.
Brain research, 986(1-2), 54-62 (2003-09-11)
The temporally protracted heredodegeneration of cerebellar Purkinje cells in shaker mutant rats can be modified: ablation of the inferior olive accelerates their degeneration whereas chronic intraventricular infusion of trophic factors extends their survival. The present study sought to determine if
M Kaffashian et al.
Physiological research, 60(2), 355-365 (2010-12-01)
Alterations in the intrinsic properties of Purkinje cells (PCs) may contribute to the abnormal motor performance observed in ataxic rats. To investigate whether such changes in the intrinsic neuronal excitability could be attributed to the role of Ca(2+)-activated K(+) channels
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