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About This Item
经验公式（希尔记法）:
Ni3S2
CAS号:
分子量:
240.21
EC號碼:
MDL號碼:
分類程式碼代碼:
12161600
PubChem物質ID:
NACRES:
NA.23
化驗
99.7% trace metals basis
形狀
powder
反應適用性
reagent type: catalyst
core: nickel
粒徑
−150 mesh
密度
5.8 g/mL at 25 °C (lit.)
應用
battery manufacturing
SMILES 字串
[Ni]=S=[Ni]=S=[Ni]
InChI
1S/3Ni.2S
InChI 密鑰
YGHCWPXPAHSSNA-UHFFFAOYSA-N
相关类别
一般說明
Major phase is Ni3S2. Product may also include a small amount of sulfur-rich phase. Nickel Sulfide (Ni3S2) exists as a heazlewoodite mineral in nature. The structure of Ni3S2 is hombohedral. Raman spectral analysis of Ni3S2 has been reported.
應用
Nickel sulfide was implanted in mice, to induce the growth of malignant fibrous histiocytomas.
包裝
Packaged in glass bottles
訊號詞
Danger
危險分類
Acute Tox. 3 Inhalation - Aquatic Acute 1 - Aquatic Chronic 1 - Carc. 1A Inhalation - Muta. 2 - Skin Sens. 1 - STOT RE 1 Inhalation
標靶器官
Lungs
儲存類別代碼
6.1D - Non-combustible acute toxic Cat.3 / toxic hazardous materials or hazardous materials causing chronic effects
水污染物質分類（WGK）
WGK 3
閃點（°F）
Not applicable
閃點（°C）
Not applicable
個人防護裝備
Eyeshields, Faceshields, Gloves, type P3 (EN 143) respirator cartridges
Reactive oxygen-induced carcinogenesis causes hypermethylation of p16 (Ink4a) and activation of MAP kinase.
Govindarajan B, et al.
Molecular Medicine, 8(1) (2002)
C Huang et al.
Cancer research, 61(22), 8051-8057 (2001-11-24)
Nickel compounds induce cell transformation in cell culture models and tumor formation in experimental animals. However, the molecular mechanisms by which nickel compounds induce tumors are not yet well understood. The present study found that exposure of cells to either
Frank E Huggins et al.
Environmental science & technology, 45(14), 6188-6195 (2011-06-30)
XAFS spectroscopy has been used to determine the Ni species in particulate matter collected on quartz thimble filters in the stacks of eight residual (No. 6 fuel) oil-burning electric utility steam-generating units. Proper speciation of nickel in emitted particulate matter
Shosuke Kawanishi et al.
Environmental health perspectives, 110 Suppl 5, 789-791 (2002-11-12)
The U.S. National Toxicology Program has shown clear evidence of carcinogenicity of nickel subsulfide (Ni(3)S(2)) and some evidence of carcinogenicity of NiO (green) in rats. In the present study, DNA damage in cultured cells and in lungs of rats induced
A S Andrew et al.
American journal of physiology. Lung cellular and molecular physiology, 281(3), L607-L615 (2001-08-16)
Human epidemiological and animal studies have associated inhalation of nickel dusts with an increased incidence of pulmonary fibrosis. At the cellular level, particulate nickel subsulfide inhibits fibrinolysis by transcriptionally inducing expression of plasminogen activator inhibitor (PAI)-1, an inhibitor of the
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