D184
Diethylamine NONOate sodium salt hydrate
≥97% (NMR), crystalline, nitrix oxide donor
동의어(들):
1,1-Diethyl-2-hydroxy-2-nitroso-hydrazine sodium, 2-(N,N-Diethylamino)-diazenolate 2-oxide sodium salt hydrate, DEA NONOate sodium salt hydrate, Diethylamine/nitric oxide complex sodium salt hydrate
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모든 사진(1)
About This Item
Linear Formula:
(CH3CH2)2N–N(N=O)O- Na+ xH2O
CAS Number:
Molecular Weight:
155.13 (anhydrous basis)
MDL number:
UNSPSC 코드:
12352116
PubChem Substance ID:
NACRES:
NA.77
추천 제품
제품명
Diethylamine NONOate sodium salt hydrate, ≥97% (NMR)
Quality Level
분석
≥97% (NMR)
양식
crystalline
저장 조건
desiccated
protect from light
색상
white
mp
45 °C (lit.)
solubility
H2O: >10 mg/mL
methanol: >25 mg/mL
배송 상태
dry ice
저장 온도
−20°C
SMILES string
O.[Na+].CCN(CC)N([O-])N=O
InChI
1S/C4H10N3O2.Na.H2O/c1-3-6(4-2)7(9)5-8;;/h3-4H2,1-2H3;;1H2/q-1;+1;
InChI key
BVCZSYKIXSAMOT-UHFFFAOYSA-N
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애플리케이션
Diethylamine NONOate sodium salt hydrate has been used as a nitric oxide donor to evaluate endothelium-independent dilation. It has also been used to determine the level of nitric oxide in cell free systems.
생화학적/생리학적 작용
Diethylamine NONOate is a complex of diethylamine with nitric oxide used to generate a controlled release of nitric oxide in solution.
Storage Class Code
11 - Combustible Solids
WGK
WGK 3
Flash Point (°F)
Not applicable
Flash Point (°C)
Not applicable
개인 보호 장비
Eyeshields, Gloves, type N95 (US)
Extracellular Alpha-Synuclein Oligomers Induce Parkin S-Nitrosylation: Relevance to Sporadic Parkinson?s Disease Etiopathology
Wilkaniec A, et al.
Molecular Neurobiology, 56(1), 125-140 (2019)
The functional and structural changes in the basilar artery due to overpressure blast injury
Toklu HZ, et al.
Journal of Cerebral Blood Flow and Metabolism, 35(12), 1950-1956 (2015)
Francesco Tamagnini et al.
The Journal of physiology, 591(16), 3963-3979 (2013-05-15)
Synaptic plasticity in perirhinal cortex is essential for recognition memory. Nitric oxide and endocannabinoids (eCBs), which are produced in the postsynaptic cell and act on the presynaptic terminal, are implicated in mechanisms of long-term potentiation (LTP) and long-term depression (LTD)
High dietary fructose does not exacerbate the detrimental consequences of high fat diet on basilar artery function
Toklu HZ, et al.
Journal of Physiology And Pharmacology, 67(2), 205-205 (2016)
Nina Jeppesen Edin et al.
Journal of radiation research, 54(6), 1015-1028 (2013-05-21)
In this study, a mechanism in which low-dose hyper-radiosensitivity (HRS) is permanently removed, induced by low-dose-rate (LDR) (0.2-0.3 Gy/h for 1 h) but not by high-dose-rate priming (0.3 Gy at 40 Gy/h) was investigated. One HRS-negative cell line (NHIK 3025)
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