추천 제품
Quality Level
분석
97%
양식
powder
bp
142 °C/125 mmHg (lit.)
mp
76-79 °C (lit.)
solubility
95% ethanol: soluble 5%, clear to very slightly hazy, colorless to light yellow
작용기
aldehyde
ketone
phenyl
SMILES string
[H]O[H].[H]C(=O)C(=O)c1ccccc1
InChI
1S/C8H6O2.H2O/c9-6-8(10)7-4-2-1-3-5-7;/h1-6H;1H2
InChI key
YQBLQKZERMAVDO-UHFFFAOYSA-N
유사한 제품을 찾으십니까? 방문 제품 비교 안내
애플리케이션
Phenylglyoxal hydrate was used :
- to modify pig muscle carbonic anhydraseIII
- as specific reagent for arginine groups
- to prepare pyrrolinone and furan derivatives
- as chemiluminescent reagent for the determination of purines
생화학적/생리학적 작용
Phenylglyoxal is a potent inhibitor of mitochondrial aldehyde dehydrogenase. It reacts with arginine residues in purified Hageman factor (HF, Factor XII) and causes inhibition of its coagulant properties.
신호어
Warning
유해 및 위험 성명서
Hazard Classifications
Acute Tox. 4 Oral - Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3
표적 기관
Respiratory system
Storage Class Code
11 - Combustible Solids
WGK
WGK 3
Flash Point (°F)
Not applicable
Flash Point (°C)
Not applicable
개인 보호 장비
dust mask type N95 (US), Eyeshields, Faceshields, Gloves
이미 열람한 고객
Analytica Chimica Acta, 287, 75-75 (1994)
O D Radnoff et al.
Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine (New York, N.Y.), 148(1), 177-182 (1975-01-01)
Exposure of purified Hageman factor (HF, Factor XII) to phenylglyoxal hydrate (PHG), an agent reacting with arginine residues in protein, inhibited its coagulant properties upon subsequent exposure of negatively charged agents. Once HF had been exposed to kaolin or ellagic
L M Pullan et al.
Biochemistry, 24(3), 635-640 (1985-01-29)
Mammalian carbonic anhydrase III has previously been shown to catalyze the hydrolysis of p-nitrophenyl phosphate in addition to possessing the conventional CO2 hydratase and p-nitrophenylacetate esterase activities. Modification of pig muscle carbonic anhydrase III with the arginine reagent phenylglyoxal yielded
William A Irwin et al.
Biochemical and biophysical research communications, 291(2), 215-219 (2002-02-16)
Fructose has been shown to protect hepatocyte viability during hypoxia or exposure to mitochondrial electron transport inhibitors. We report here that the fructose metabolite D-glyceraldehyde (D-GA) is a good inhibitor of the mitochondrial permeability transition pore (PTP) in isolated rat
Synthesis, 783-783 (1993)
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