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GENA935

ECL Rat IgG, HRP-linked whole antibody (from goat)

Cytiva NA935

Synonym(s):

Rat IgG antibody

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About This Item

UNSPSC Code:
12352203
NACRES:
NA.46

antibody form

Ig fraction of antiserum

antibody product type

primary antibodies

feature

wetted part: no

packaging

pkg of 1 mL

manufacturer/tradename

Cytiva NA935

color

Brown (light)

shipped in

wet ice

storage temp.

2-8°C

General description

The antibody is prepared by hyper-immunizing goats with purified immunoglobulin fractions from normal rat serum to produce high affinity antibodies. The pooled antiserum is used to produce an immunoglobulin preparation which is then affinity adsorbed to remove cross-reacting antibodies towards mouse, human and rabbit immunoglobulins. These activities are thoroughly depleted to ensure species-specificity. Finally, to select for specific binding to rat IgG, the antibodies are purified using an affinity column of rat IgG. After washing to remove non-specific serum components and low affinity antibodies, the species-specific antibodies are eluted using carefully selected, mild conditions which minimize aggregation and preserve immunological activity, yet which will elute high affinity antibodies.

Features and Benefits

  • Highly species-specific.
  • Optimized for use with our range of ECL Detection Reagents.
  • Recommended dilutions to minimize background.

Storage and Stability

Please be aware this product may be shipped 90 days before the expiration date. For more information on the batch specific expiration date, please contact technical service.

Analysis Note

To view the Certificate of Analysis for this product, please visit www.cytiva.com.

Legal Information

ECL is a trademark of Cytiva

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Hazard Statements

Storage Class Code

12 - Non Combustible Liquids


Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Macroautophagy/autophagy failure with the accumulation of autophagosomes is an early neuropathological feature of Alzheimer disease (AD) that directly affects amyloid beta (Aβ) metabolism. Although loss of presenilin 1 function has been reported to impair lysosomal function and prevent autophagy flux

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