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Merck
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Key Documents

T180

Sigma-Aldrich

NK2 Tachykinin Receptor human

recombinant, expressed in CHO cells, membrane suspension

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About This Item

MDL號碼:
分類程式碼代碼:
12352200

重組細胞

expressed in CHO cells

品質等級

形狀

membrane suspension

包裝

vial of 50 units

UniProt登錄號

運輸包裝

dry ice

儲存溫度

−70°C

基因資訊

human ... TACR2(6865)

外觀

Supplied in 50 mM Tris-HCl, pH 7.4, containing 10% glycerol and 1% BSA. Actual concentrations and specific binding capacity are provided with each lot.

分析報告

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X P Zeng et al.
The Journal of urology, 153(5), 1688-1692 (1995-05-01)
Functional and radioligand binding studies with selective agonists and antagonists were used to investigate tachykinin receptors in the human bladder. Strips of detrusor muscle were contracted by the tachykinins neurokinin A and neuropeptide gamma, and by the NK2 receptor selective
Daolin Tang et al.
American journal of respiratory cell and molecular biology, 41(6), 651-660 (2009-03-07)
The pathogenesis of sepsis is mediated in part by the pathogen-associated molecular pattern molecule bacterial endotoxin, which stimulates macrophages to sequentially release early (e.g., TNF-alpha, IL-1beta) and late (e.g., high-mobility group box [HMGB] 1 protein) proinflammatory mediators. The recent discovery
Long Sun et al.
Molecular medicine reports, 17(2), 3274-3280 (2017-12-20)
The purpose of the present study was to examine the effects of myricetin on reducing cerebral ischemia injury in a rat model. A rat model of permanent middle cerebral artery occlusion (pMCAO) was used in the present study. Rats were
Josefine Stockert et al.
PloS one, 8(4), e63011-e63011 (2013-05-07)
The peroxisome proliferator-activated receptor subtypes PPARα, PPARβ/δ, PPARγ are members of the steroid hormone receptor superfamily with well-established functions in transcriptional regulation. Here, we describe an unexpected cytoplasmic function of PPARβ/δ. Silencing of PPARβ/δ expression interferes with the expression of
Benjamin R Pryce et al.
Biochimica et biophysica acta. Molecular cell research, 1868(2), 118917-118917 (2020-12-02)
Duchenne's muscular dystrophy (DMD) is a severe muscle wasting disorder characterized by the loss of dystrophin expression, muscle necrosis, inflammation and fibrosis. Ongoing muscle regeneration is impaired by persistent cytokine stress, further decreasing muscle function. Patients with DMD rarely survive

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