推荐产品
品質等級
化驗
≥98% (HPLC)
形狀
solid
藥物控制
USDEA Schedule IV
顏色
white
溶解度
DMSO: >10 mg/mL
起源
Teva
SMILES 字串
NC(=O)CS(=O)C(c1ccccc1)c2ccccc2
InChI
1S/C15H15NO2S/c16-14(17)11-19(18)15(12-7-3-1-4-8-12)13-9-5-2-6-10-13/h1-10,15H,11H2,(H2,16,17)
InChI 密鑰
YFGHCGITMMYXAQ-UHFFFAOYSA-N
基因資訊
human ... SLC6A3(6531)
rat ... Slc6a3(24898)
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生化/生理作用
Modafinil is a central nervous system vigilance promoting agent, which posseses neuroprotective properties.
特點和優勢
This compound was developed by Teva. To browse the list of other pharma-developed compounds and Approved Drugs/Drug Candidates, click here.
訊號詞
Warning
危險聲明
危險分類
Acute Tox. 4 Oral - Repr. 2
儲存類別代碼
11 - Combustible Solids
水污染物質分類(WGK)
WGK 3
閃點(°F)
Not applicable
閃點(°C)
Not applicable
個人防護裝備
Eyeshields, Gloves, type N95 (US)
Scientific reports, 11(1), 1843-1843 (2021-01-21)
At high latitudes, approximately 10% of people suffer from depression during the winter season, a phenomenon known as seasonal affective disorder (SAD). Shortened photoperiod and/or light intensity during winter season are risk factors for SAD, and bright light therapy is
British journal of clinical pharmacology, 75(3), 728-737 (2012-10-10)
Over recent years there has been increasing research into both pharmaceutical and nutraceutical cognition enhancers. Here we aimed to calculate the effect sizes of positive cognitive effect of the pharmaceutical modafinil in order to benchmark the effect of two widely
Cognitive and behavioral neurology : official journal of the Society for Behavioral and Cognitive Neurology, 25(3), 107-114 (2012-09-11)
Examine the efficacy of armodafinil in improving cognition in patients with multiple sclerosis (MS). Many patients with MS experience cognitive difficulties. Armodafinil has shown promise as a cognitive enhancer in other patient populations. No studies have examined whether armodafinil improves
Modafinil #259.
Journal of palliative medicine, 15(12), 1388-1389 (2012-12-05)
PloS one, 7(9), e45471-e45471 (2012-10-03)
We have previously shown that modafinil promotes wakefulness via dopamine receptor D(1) and D(2) receptors; however, the locus where dopamine acts has not been identified. We proposed that the nucleus accumbens (NAc) that receives the ventral tegmental area dopamine inputs
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