推荐产品
形狀
solution
儲存期限
Expiry date on the label
IVD
for in vitro diagnostic use
濃度
2.3%
技術
microbe id | staining: suitable
pH值
4.6
應用
hematology
histology
儲存溫度
room temp
SMILES 字串
[Cl-].CN(C)c1ccc(cc1)\C(c2ccc(cc2)N(C)C)=C3/C=C\C(C=C3)=[N+](/C)C
InChI
1S/C25H30N3.ClH/c1-26(2)22-13-7-19(8-14-22)25(20-9-15-23(16-10-20)27(3)4)21-11-17-24(18-12-21)28(5)6;/h7-18H,1-6H3;1H/q+1;/p-1
InChI 密鑰
ZXJXZNDDNMQXFV-UHFFFAOYSA-M
正在寻找类似产品? 访问 产品对比指南
應用
用于革兰氏染色方案HT90(Gram Stain Procedure No. HT90)。
成分
含有2.3%认证的结晶紫、0.1%草酸铵和20%乙醇(SD3A变性酒精)。
訊號詞
Warning
危險分類
Aquatic Chronic 3 - Carc. 2 - Eye Irrit. 2 - Flam. Liq. 3
儲存類別代碼
3 - Flammable liquids
水污染物質分類（WGK）
WGK 2
閃點（°F）
100.0 °F - closed cup
閃點（°C）
37.8 °C - closed cup
個人防護裝備
Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter
其他客户在看
Chia-Hsun Fang et al.
Journal of biomedical science, 27(1), 42-42 (2020-03-15)
The underlying mechanism involved in ovarian cancer stemness and chemoresistance remains largely unknown. Here, we explored whether the regulation of c-Kit and plasma membrane prohibitin (PHB) affects ovarian cancer stemness and chemotherapy resistance. Mass spectrum analysis and an in vitro
Bin Xiao et al.
Cell reports, 31(4), 107571-107571 (2020-04-30)
Mechanistic target of rapamycin complex 1 (mTORC1) is a master modulator of cellular growth, and its aberrant regulation is recurrently documented within breast cancer. While the small GTPase Rheb1 is the canonical activator of mTORC1, Rheb1-independent mechanisms of mTORC1 activation
Daniel Haensel et al.
EMBO reports, 20(1) (2018-11-11)
Directional migration is inherently important for epithelial tissue regeneration and repair, but how it is precisely controlled and coordinated with cell proliferation is unclear. Here, we report that Ovol2, a transcriptional repressor that inhibits epithelial-to-mesenchymal transition (EMT), plays a crucial
Chao Wan et al.
Science advances, 6(13), eaay9789-eaay9789 (2020-04-02)
Radiotherapy (RT) is routinely used in cancer treatment, but expansion of its clinical indications remains challenging. The mechanism underlying the radiation-induced bystander effect (RIBE) is not understood and not therapeutically exploited. We suggest that the RIBE is predominantly mediated by
Niklas Gremke et al.
Nature communications, 11(1), 4684-4684 (2020-09-19)
Cancer cells have a characteristic metabolism, mostly caused by alterations in signal transduction networks rather than mutations in metabolic enzymes. For metabolic drugs to be cancer-selective, signaling alterations need to be identified that confer a druggable vulnerability. Here, we demonstrate
我们的科学家团队拥有各种研究领域经验，包括生命科学、材料科学、化学合成、色谱、分析及许多其他领域.
联系客户支持