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Merck

M8640

Sigma-Aldrich

1-甲氧基-5-甲基吩嗪甲硫酸盐

≥95% purity , powder

别名:

1-甲氧基吩嗪甲硫酸酯

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About This Item

经验公式(希尔记法):
C15H16N2O5S
CAS号:
分子量:
336.36
EC號碼:
MDL號碼:
分類程式碼代碼:
12171500
PubChem物質ID:
NACRES:
NA.47

product name

1-甲氧基-5-甲基吩嗪甲硫酸盐, ≥95%

品質等級

化驗

≥95%

形狀

powder

成份

Nitrogen: ≥ 7.9%

技術

titration: suitable

顏色

red to brown

溶解度

H2O: 1 mg/mL, clear, red

應用

diagnostic assay manufacturing
hematology
histology

儲存溫度

room temp

SMILES 字串

COS([O-])(=O)=O.COc1cccc2[n+](C)c3ccccc3nc12

InChI

1S/C14H13N2O.CH4O4S/c1-16-11-7-4-3-6-10(11)15-14-12(16)8-5-9-13(14)17-2;1-5-6(2,3)4/h3-9H,1-2H3;1H3,(H,2,3,4)/q+1;/p-1

InChI 密鑰

MASUWVVNWALEEM-UHFFFAOYSA-M

一般說明

1-甲氧基-5-甲基吩嗪甲基硫酸盐(MPMS)是NAD (P) H与四氮唑染料间的一种电化学稳定的电子传递介质。MPMS的还原形式是将四唑盐转化为紫色沉淀甲。它是一种非常稳定的非酶电子载体,在体外介导脱氢酶向各种电子受体的电子转移,比如四氮唑染料或酶电池的电极等。

應用

1-甲氧基-5-甲基吩嗪硫酸甲酯已被用作缓冲液或溶液的成分,用于乳酸脱氢酶和肌肉琥珀酸脱氢酶测定。

象形圖

Health hazardExclamation mark

訊號詞

Warning

危險分類

Acute Tox. 4 Oral - Carc. 2 - Eye Irrit. 2 - Muta. 2 - Repr. 2 - Skin Irrit. 2 - STOT RE 2

標靶器官

Nervous system

儲存類別代碼

11 - Combustible Solids

水污染物質分類(WGK)

WGK 3

個人防護裝備

Eyeshields, Gloves, type P3 (EN 143) respirator cartridges


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Early life exposure to chronic intermittent hypoxia causes upper airway dilator muscle weakness, which persists into young adulthood.
McDonald FB, et. al.
Experimental Physiology, 100, 947-947 (2015)
Kuniyo Inouye et al.
Journal of biochemistry, 131(1), 97-105 (2002-01-05)
A synchronous enzyme-reaction system using water-soluble formazan and a non-enzymatic electron mediator was developed and applied to an enzyme immunoassay (EIA). The reaction system consists of four steps: (I) dephosphorylation of NADP(+) to produce NAD(+) by alkaline phosphatase (ALP), (II)
Early life exposure to chronic intermittent hypoxia causes upper airway dilator muscle weakness, which persists into young adulthood
McDonald FB, et al.
Experimental Physiology, 100(8), 947-966 (2015)
Patries M Herst et al.
Biochimica et biophysica acta, 1767(2), 170-177 (2007-02-03)
Oxygen consumption for bioenergetic purposes has long been thought to be the prerogative of mitochondria. Nevertheless, mitochondrial gene knockout (rho(0)) cells that are defective in mitochondrial respiration require oxygen for growth and consume oxygen at the cell surface via trans-plasma
Methods in Plant Cell Biology (1995)

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