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Merck
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Key Documents

ST1643

Sigma-Aldrich

Anti-PDK2 Mouse mAb (2G1)

liquid, clone 2G1, Calbiochem®

别名:

Anti-Pyruvate Dehydrogenase Kinase 2

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About This Item

分類程式碼代碼:
12352203
NACRES:
NA.41

生物源

mouse

品質等級

抗體表格

purified antibody

抗體產品種類

primary antibodies

無性繁殖

2G1, monoclonal

形狀

liquid

不包含

preservative

物種活性

human

製造商/商標名

Calbiochem®

儲存條件

OK to freeze
avoid repeated freeze/thaw cycles

同型

IgG2a

運輸包裝

wet ice

儲存溫度

−20°C

目標翻譯後修改

unmodified

基因資訊

human ... PDK2(5164)

一般說明

Purified mouse monoclonal antibody. Recognizes the ~40 kDa PDK2 protein.
Recognizes the ~40 kDa PDK2 protein in U-2 OS cells.
This Anti-PDK2 Mouse mAb (2G1) is validated for use in ELISA, Immunoblotting, Paraffin Sections for the detection of PDK2.

免疫原

A recombinant polypeptide corresponding to amino acids of 187-276 human PDK2, expressed as a GST fusion protein

應用



ELISA (see comments)
Immunoblotting (1-5 g/ml)
Paraffin Sections (0.8 g/ml)

警告

Toxicity: Regulatory Review (Z)

外觀

In PBS, pH 7.2.

重構

Following initial thaw, aliquot and freeze (-20°C).

分析報告

Negative Control
293T cells
Positive Control
U-2 OS cells

其他說明

For ELISA, this antibody can be used as the capture antibody; recommended concentration is 10 ng/ml, 1 µg/well, 100 µl total volume. Variables associated with assay conditions will dictate the proper working dilution.

法律資訊

CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany

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儲存類別代碼

12 - Non Combustible Liquids

水污染物質分類(WGK)

nwg

閃點(°F)

Not applicable

閃點(°C)

Not applicable


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Anders Gudiksen et al.
Physiological reports, 5(7) (2017-04-13)
The purpose of the present study was to examine whether lack of skeletal muscle peroxisome proliferator-activated receptor gamma coactivator 1 alpha (PGC-1α) affects the switch in substrate utilization from a fed to fasted state and the fasting-induced pyruvate dehydrogenase (PDH)
Jakob G Knudsen et al.
Physiological reports, 4(9) (2016-05-18)
Interleukin (IL)-6 is released from skeletal muscle (SkM) during exercise and has been shown to affect hepatic metabolism. It is, however, unknown whether SkM IL-6 is involved in the regulation of exercise training-induced counteraction of changes in carbohydrate and lipid

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