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形狀
powder
包裝
pkg of 1 × 5 mg (870714P-5mg)
製造商／商標名
Avanti Polar Lipids 870714P
應用
lipidomics
脂質類型
coenzymes
運輸包裝
dry ice
儲存溫度
−20°C
SMILES 字串
O[C@@](C(NCCC(NCCSC(CCCCCCCCCCCCC)=O)=O)=O)(C(C)(COP([O-])(OP([O-])(OC[C@H]([C@H]1OP([O-])(O)=O)O[C@H]([C@@H]1O)N2C3=C(C(N)=NC=N3)N=C2)=O)=O)C)[H].[NH4+].[NH4+].[NH4+]
InChI
1S/C35H62N7O17P3S.3H3N/c1-4-5-6-7-8-9-10-11-12-13-14-15-26(44)63-19-18-37-25(43)16-17-38-33(47)30(46)35(2,3)21-56-62(53,54)59-61(51,52)55-20-24-29(58-60(48,49)50)28(45)34(57-24)42-23-41-27-31(36)39-22-40-32(27)42;;;/h22-24,28-30,34,45-46H,4-21H2,1-3H3,(H,37,43)(H,38,47)(H,51,52)(H,53,54)(H2,36,39,40)(H2,48,49,50);3*1H3/t24-,28?,29+,30+,34-;;;/m1.../s1
InChI 密鑰
DSAPDQDCGNIALG-YNLIGXKZSA-N
一般說明
14:0 Coenzyme A, also known as myristoyl coenzyme A, is a coenzyme A derivative of myristic acid. 14:0 Coenzyme A is a long-chain acyl CoA.
應用
14:0 Coenzyme A may be used as a standard in quantitative ultraperformance liquid chromatography-tandem mass spectrometry (UPLC-MS/MS) method for simultaneous analysis of tissue long-chain acyl-CoA (LCACoA) concentration.
生化／生理作用
14:0 Coenzyme A is an important lipid metabolite. It acts as a N-myristoyltransferase substrate during myristoylation. Myristoylation is a protein lipid modification involving the addition of myristoyl group to proteins either during translation to modify protein activity or post-translationally in apoptotic cells.
包裝
5 mL Amber Glass Screw Cap Vial (870714P-5mg)
法律資訊
Avanti Research is a trademark of Avanti Polar Lipids, LLC
儲存類別代碼
11 - Combustible Solids
R S Bhatnagar et al.
Biochimica et biophysica acta, 1441(2-3), 162-172 (1999-11-26)
Protein N-myristoylation is a covalent modification that occurs co-translationally in eukaryotes. Myristate, a rare 14 carbon saturated fatty acid (C14:0), is attached, via an amide linkage, to the N-terminal glycine of a subset of eukaryotic and viral proteins by myristoyl-CoA:protein
Agnieszka U Blachnio-Zabielska et al.
Rapid communications in mass spectrometry : RCM, 25(15), 2223-2230 (2011-07-08)
Long-chain acyl-coenzymes A (acyl-CoAs) (LCACoA) are the activated forms of long-chain fatty acids and serve as key lipid metabolites. Excess accumulation of intracellular LCACoA, diacylglycerols (DAGs) and ceramides may create insulin resistance with respect to glucose metabolism. We present a
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