推荐产品
生物源
synthetic
等級
Halal
濃度
40 wt. % in H2O
折射率
n20/D 1.41
密度
1.178 g/mL at 25 °C
應用
flavors and fragrances
文件
see Safety & Documentation for available documents
食物過敏原
no known allergens
感官的
caramel
儲存溫度
2-8°C
SMILES 字串
[H]C(=O)C(C)=O
InChI
1S/C3H4O2/c1-3(5)2-4/h2H,1H3
InChI 密鑰
AIJULSRZWUXGPQ-UHFFFAOYSA-N
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一般說明
丙酮醛溶液可与铵盐一起用于制备模拟大气气溶胶成分的水溶液，以研究可吸收次级有机材料的形成。
免責聲明
仅供R&D或非EU食品使用不用于零售。
訊號詞
Danger
危險分類
Eye Dam. 1 - Met. Corr. 1 - Muta. 2 - Skin Sens. 1
儲存類別代碼
8A - Combustible corrosive hazardous materials
水污染物質分類（WGK）
WGK 1
閃點（°F）
Not applicable
閃點（°C）
Not applicable
個人防護裝備
Eyeshields, Faceshields, Gloves, type ABEK (EN14387) respirator filter
其他客户在看
Secondary organic material formed by methylglyoxal in aqueous aerosol mimics.
Sareen N, et al.
Atmospheric Chemistry and Physics, 10(3) (2010)
T W Lo et al.
Biochemical pharmacology, 48(10), 1865-1870 (1994-11-16)
Increased formation of methylglyoxal in clinical diabetes mellitus and metabolism by the glyoxalase system has been linked to the development of clinical complications of diabetes: retinopathy, neuropathy and nephropathy. Aminoguanidine has been proposed as a prophylactic agent for preventive therapy
Georg T Wondrak et al.
Biochemical pharmacology, 63(3), 361-373 (2002-02-21)
Tissue deterioration and aging have long been associated with the accumulation of chemically induced protein and DNA damage. Reactive oxygen species (ROS) and reactive carbonyl species (RCS), especially alpha-dicarbonyl compounds, are key mediators of damage caused by oxidative stress, glycation
Wei-Hsuan Hsu et al.
Toxicology and applied pharmacology, 272(3), 842-851 (2013-08-21)
Methylglyoxal (MG) is a toxic-glucose metabolite and a major precursor of advanced glycation endproducts (AGEs). MG has been reported to result in inflammation by activating receptor for AGEs (RAGE). We recently found that Monascus-fermented metabolite monascin acts as a novel
Paul J Beisswenger et al.
Diabetes, 54(11), 3274-3281 (2005-10-27)
Dicarbonyl and oxidative stress may play important roles in the development of diabetes complications, and their response to hyperglycemia could determine individual susceptibility to diabetic nephropathy. This study examines the relationship of methylglyoxal, 3-deoxyglucosone (3DG), and oxidative stress levels to
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