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Key Documents

SRP4560

Sigma-Aldrich

Resistin from mouse

recombinant, expressed in E. coli, ≥98% (SDS-PAGE), ≥98% (HPLC)

Sinónimos:

ADSF, Adipose tissue-specific secretory factor, C/EBP-ε-regulated myeloid-specific secreted cysteine-rich protein, Cysteine-rich secreted protein A12-α-like 2, Cysteine-rich secreted protein FIZZ3, RSTN

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About This Item

Número MDL:
Código UNSPSC:
12352200
NACRES:
NA.32

origen biológico

mouse

recombinante

expressed in E. coli

Análisis

≥98% (HPLC)
≥98% (SDS-PAGE)

formulario

lyophilized

mol peso

~20.6 kDa

envase

pkg of 25 μg

impurezas

endotoxin, tested

Nº de acceso NCBI

Condiciones de envío

wet ice

temp. de almacenamiento

−20°C

Información sobre el gen

mouse ... Retn(57264)

Descripción general

Mouse Resistin is a new member to the family of adipocyte secreted proteins. The biological functions of Resistin as well as its molecular target are largely unknown. Whether it promotes or antagonizes insulin action is still unclear. Recombinant mouse Resistin is a 20.2 kDa dimeric protein consisting of two 94 amino acid polypeptide chains.

Forma física

Lyophilized from 20 mM Tris, pH 8.0.

Reconstitución

Centrifuge the vial prior to opening. Avoid freeze-thaw cycles.
Reconstitution in water to a concentration of 0.1-1.0 mg/mL. This solution can then be diluted into other aqueous buffers.

Código de clase de almacenamiento

13 - Non Combustible Solids

Clase de riesgo para el agua (WGK)

nwg

Punto de inflamabilidad (°F)

Not applicable

Punto de inflamabilidad (°C)

Not applicable


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Fengyun Wen et al.
Biochemical genetics, 53(4-6), 63-71 (2015-05-13)
Resistin is associated with ectopic deposition of lipids, and determining its developmental and molecular mechanisms may help in the development of novel treatments. MicroRNAs (miRNAs) are involved in many physiological and pathological processes as negative regulators. We performed mouse liver
Jiawen Yong et al.
Journal of cellular and molecular medicine, 25(20), 9710-9723 (2021-09-16)
Hypoxia-induced apoptosis of cementoblasts (OCCM-30) may be harmful to orthodontic treatment. Hypoxia-inducible factor 1-alpha (HIF-1α) mediates the biological effects during hypoxia. Little is known about the survival mechanism capable to counteract cementoblast apoptosis. We aimed to investigate the potential roles
Chen-Ming Su et al.
Stem cells (Dayton, Ohio), 33(7), 2243-2255 (2015-04-02)
Endothelial progenitor cells (EPCs) promote angiogenesis and are therefore key contributors to a wide variety of angiogenesis-related autoimmune diseases such as rheumatoid arthritis (RA). However, the signaling mechanisms through which these progenitor cells influence RA pathogenesis remain unknown. The aim

Artículos

Lipid Induced Insulin Resistance

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