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Key Documents

Altri documenti

05164

Supelco

(Aminomethyl)phosphonic acid

PESTANAL®, analytical standard

Sinonimo/i:

AMPA

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About This Item

Formula condensata:
NH2CH2P(O)(OH)2
Numero CAS:
1066-51-9
Peso molecolare:
111.04
Beilstein:
1701219
Numero MDL:
MFCD00008105
Codice UNSPSC:
77101502
ID PubChem:
329747650
NACRES:
NA.24

Grado

analytical standard

Livello qualitativo

100

Nome Commerciale

PESTANAL®

Saggio

≥98.0% (TLC)

Durata

limited shelf life, expiry date on the label

Impurezze

≤3.0% water (calc. from elemental analysis)

applicazioni

agriculture
environmental

Formato

neat

Stringa SMILE

NCP(O)(O)=O

InChI

1S/CH6NO3P/c2-1-6(3,4)5/h1-2H2,(H2,3,4,5)
MGRVRXRGTBOSHW-UHFFFAOYSA-N

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Find a digital Reference Material for this product available on our online platform ChemisTwin® for NMR. You can use this digital equivalent on ChemisTwin® for your sample identity confirmation and compound quantification (with digital external standard). An NMR spectrum of this substance can be viewed and an online comparison against your sample can be performed with a few mouseclicks. Learn more here and start your free trial.

Note legali

PESTANAL is a registered trademark of Merck KGaA, Darmstadt, Germany

Codice della classe di stoccaggio

11 - Combustible Solids

Classe di pericolosità dell'acqua (WGK)

WGK 3

Punto d’infiammabilità (°F)

Not applicable

Punto d’infiammabilità (°C)

Not applicable

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Certificati d'analisi (COA)

Lot/Batch Number
BCCK3466
BCCG4507
BCCG4869
BCCG4198
BCCF9469

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Glufosinate-ammonium certified reference material, TraceCERT®, Manufactured by: Sigma-Aldrich Production GmbH, Switzerland

Supelco

49677

Glufosinate-ammonium

3-(Methylphosphinico)propionic acid PESTANAL®, analytical standard

Supelco

31264

3-(Methylphosphinico)propionic acid

Glifosato-2-13C,15N 98 atom % 15N, 99 atom % 13C

Sigma-Aldrich

608629

Glifosato-2-13C,15N

2-Aminoethylphosphonic acid 99%

Sigma-Aldrich

268674

2-Aminoethylphosphonic acid

4-Aminobutylphosphonic acid ≥99%

Sigma-Aldrich

A0664

4-Aminobutylphosphonic acid

Seonil Kim et al.
Proceedings of the National Academy of Sciences of the United States of America, 112(10), 3122-3127 (2015-02-26)
Gene knockout (KO) does not always result in phenotypic changes, possibly due to mechanisms of functional compensation. We have studied mice lacking cGMP-dependent kinase II (cGKII), which phosphorylates GluA1, a subunit of AMPA receptors (AMPARs), and promotes hippocampal long-term potentiation
Mary L Huff et al.
Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology, 40(4), 861-873 (2014-09-27)
Recent findings suggest that the mesolimbic dopamine neurons, known to promote cocaine-seeking behavior, are strongly inhibited by a newly characterized region of the midbrain known as the rostromedial tegmental nucleus (RMTg). The RMTg appears to be involved in generating reward-prediction
Philip Jean-Richard Dit Bressel et al.
PloS one, 9(11), e111699-e111699 (2014-11-05)
The lateral habenula (LHb) is a small epithalamic structure that projects via the fasciculus retroflexus to the midbrain. The LHb is known to modulate midbrain dopamine (DA) neurons, including inhibition of ventral tegmental area (VTA) neurons via glutamatergic excitation of
W Zhou et al.
European psychiatry : the journal of the Association of European Psychiatrists, 29(7), 419-423 (2013-12-11)
Ketamine exerts fast acting, robust, and lasting antidepressant effects in a sub-anesthetic dose, however, the underlying mechanisms are still not fully elucidated. Recent studies have suggested that ketamine's antidepressant effects are probably attributed to the activation of α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA)
Einar Ö Einarsson et al.
Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology, 40(2), 480-487 (2014-08-06)
After acquisition, hippocampus-dependent memories undergo a systems consolidation process, during which they become independent of the hippocampus and dependent on the anterior cingulate cortex (ACC) for memory expression. However, consolidated remote memories can become transiently hippocampus-dependent again following memory reactivation.
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