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Key Documents

AP106C

Sigma-Aldrich

Rabbit Anti-Goat IgG Antibody, Cy3 conjugate

Chemicon®, from rabbit

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About This Item

UNSPSC-Code:
12352203
eCl@ss:
32160702
NACRES:
NA.46

Biologische Quelle

rabbit

Qualitätsniveau

Konjugat

CY3 conjugate

Antikörperform

F(ab′)2 fragment of affinity isolated antibody

Antikörper-Produkttyp

secondary antibodies

Klon

polyclonal

Speziesreaktivität

goat

Hersteller/Markenname

Chemicon®

Methode(n)

immunofluorescence: suitable

Versandbedingung

wet ice

Posttranslationale Modifikation Target

unmodified

Anwendung

Research Category
Sekundär- & Kontrollantikörper
Research Sub Category
Sekundärantikörper gegen Gesamtimmunglobulin
This Rabbit anti-Goat IgG Antibody, Cy3 conjugate is validated for use in IF for the detection of Goat IgG.

Rechtliche Hinweise

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Haftungsausschluss

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Piktogramme

Exclamation mark

Signalwort

Warning

Gefahreneinstufungen

Aquatic Chronic 3 - Eye Irrit. 2 - Skin Irrit. 2

Lagerklassenschlüssel

11 - Combustible Solids

WGK

WGK 3


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Yiyu Deng et al.
PloS one, 9(1), e87420-e87420 (2014-02-06)
Hypoxic exposure in the perinatal period causes periventricular white matter damage (PWMD), a condition associated with myelination abnormalities. Under hypoxic conditions, glial cells were activated and released a large number of inflammatory mediators in the PWM in neonatal brain, which
Vivian W Tang
Biology direct, 1, 37-37 (2006-12-13)
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Avtar S Meena et al.
Cell reports, 39(11), 110937-110937 (2022-06-16)
Intestinal epithelial tight junction disruption is a primary contributing factor in alcohol-associated endotoxemia, systemic inflammation, and multiple organ damage. Ethanol and acetaldehyde disrupt tight junctions by elevating intracellular Ca2+. Here we identify TRPV6, a Ca2+-permeable channel, as responsible for alcohol-induced

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