AB9209
Anti-Metabotropic Glutamate Receptor 2 Antibody
Chemicon®, from rabbit
Sinônimo(s):
mGluR2
Faça loginpara ver os preços organizacionais e de contrato
About This Item
Código UNSPSC:
12352203
eCl@ss:
32160702
NACRES:
NA.41
fonte biológica
rabbit
Nível de qualidade
forma do anticorpo
affinity purified immunoglobulin
tipo de produto de anticorpo
primary antibodies
clone
polyclonal
purificado por
affinity chromatography
reatividade de espécies
human
fabricante/nome comercial
Chemicon®
técnica(s)
immunocytochemistry: suitable
immunohistochemistry: suitable (paraffin)
western blot: suitable
nº de adesão NCBI
nº de adesão UniProt
Condições de expedição
dry ice
modificação pós-traducional do alvo
unmodified
Informações sobre genes
human ... GRM2(2912)
Especificidade
Recognizes Metabotropic Glutamate Receptor 2 [mGluR2].
Imunogênio
Synthetic peptide from the N-terminal extracellular domain of human Metabotropic Glutamate Receptor 2. The immunogen has 100% conservation with rat.
Aplicação
Anti-Metabotropic Glutamate Receptor 2 Antibody detects level of Metabotropic Glutamate Receptor 2 & has been published & validated for use in IC, IH(P) & WB.
Research Category
Neuroscience
Neuroscience
Research Sub Category
Neurotransmitters & Receptors
Neurotransmitters & Receptors
Western blot
Immunohistochemistry on formalin fixed paraffin embedded tissue sections.
Immunocytochemistry on acetone fixed cells.
Optimal working dilutions must be determined by the end user.
Immunohistochemistry on formalin fixed paraffin embedded tissue sections.
Immunocytochemistry on acetone fixed cells.
Optimal working dilutions must be determined by the end user.
forma física
Affinity purified immunoglobulin. Liquid in PBS, pH 7.7 with 0.01% sodium azide.
Armazenamento e estabilidade
Maintain at -70°C in undiluted aliquots for up to 6 months after date of receipt. Avoid repeated freeze/thaw cycles.
Outras notas
Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.
Informações legais
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
Exoneração de responsabilidade
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
Não está encontrando o produto certo?
Experimente o nosso Ferramenta de seleção de produtos.
Código de classe de armazenamento
10 - Combustible liquids
Classe de risco de água (WGK)
WGK 2
Ponto de fulgor (°F)
Not applicable
Ponto de fulgor (°C)
Not applicable
Certificados de análise (COA)
Busque Certificados de análise (COA) digitando o Número do Lote do produto. Os números de lote e remessa podem ser encontrados no rótulo de um produto após a palavra “Lot” ou “Batch”.
Já possui este produto?
Encontre a documentação dos produtos que você adquiriu recentemente na biblioteca de documentos.
B Á Nic Dhonnchadha et al.
Behavioural brain research, 234(1), 100-106 (2012-06-23)
Extinguishing abnormally strengthened learned responses to cues associated with drugs of abuse remains a key tactic for alleviating addiction. To assist in developing pharmacotherapies to augment exposure therapy for relapse prevention, investigation into neurobiological underpinnings of drug-cue extinction learning is
Won-Mee Park et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 31(16), 5909-5920 (2011-04-22)
Coupling of neurons by electrical synapses (gap junctions) transiently increases in the mammalian CNS during development. We report here that the developmental increase in neuronal gap junction coupling and expression of connexin 36 (Cx36; neuronal gap junction protein) are regulated
Yongfu Wang et al.
The Journal of neuroscience : the official journal of the Society for Neuroscience, 32(2), 713-725 (2012-01-13)
In the mammalian CNS, excessive release of glutamate and overactivation of glutamate receptors are responsible for the secondary (delayed) neuronal death following neuronal injury, including ischemia, traumatic brain injury (TBI), and epilepsy. The coupling of neurons by gap junctions (electrical
Haopeng Zhang et al.
Hippocampus, 25(11), 1299-1313 (2015-03-06)
Whereas the ionotropic glutamate receptors are the major mediator in glutamatergic transmission, the metabotropic glutamate receptors (mGluRs) usually play a modulatory role. Whereas the entorhinal cortex (EC) is an essential structure involved in the generation and propagation of epilepsy, the
Nossa equipe de cientistas tem experiência em todas as áreas de pesquisa, incluindo Life Sciences, ciência de materiais, síntese química, cromatografia, química analítica e muitas outras.
Entre em contato com a assistência técnica