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Key Documents

L7527

Sigma-Aldrich

Lipoprotein, very low density from human plasma

≥95% (SDS-PAGE), solution

Synonyme(s) :

Pre-β-lipoprotein, VLDL, Very low density lipoprotein

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About This Item

Numéro CAS:
308068-14-6
Numéro MDL:
MFCD00164409
Code UNSPSC :
12352211
Nomenclature NACRES :
NA.25

Source biologique

human plasma

Niveau de qualité

200

Pureté

≥95% (SDS-PAGE)

Forme

solution

Numéro d'accès UniProt

P02649
P02655
P04114

Groupe fonctionnel

ester
phospholipid

Conditions d'expédition

wet ice

Température de stockage

2-8°C

Informations sur le gène

human ... APOB(338) , APOC2(344) , APOE(348)

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Actions biochimiques/physiologiques

VLDL (Very low density lipoprotein) may be used to study the mechanisms and effects of very low density receptors (VLDLR). VLDL is used as a potential source of fatty acids that support cardiac energy metabolism. VLDL is often studied to differentiate its effects from those of chylomicrons.
Inhibits DNA synthesis in lymphocytes activated by the nonspecific mitogen concanavalin A (Con A).

Forme physique

Solution in 150 mM NaCl and 0.01% EDTA, pH 7.4

Autres remarques

View more information on lipoprotein function and lipid transport at www.sigma-aldrich.com/enzymeexplorer

Code de la classe de stockage

12 - Non Combustible Liquids

Classe de danger pour l'eau (WGK)

WGK 2

Point d'éclair (°F)

Not applicable

Point d'éclair (°C)

Not applicable

Certificats d'analyse (COA)

Recherchez un Certificats d'analyse (COA) en saisissant le numéro de lot du produit. Les numéros de lot figurent sur l'étiquette du produit après les mots "Lot" ou "Batch".

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Resistin-like molecule (RELM) β is a secretory protein homologous to resistin and reportedly contributes to local immune response regulation in gut and bronchial epithelial cells. However, we found that activated macrophages also express RELMβ and thus investigated the role of
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Since Zilversmit first proposed postprandial lipemia as the most common risk of cardiovascular disease, chylomicrons (CM) and CM remnants have been thought to be the major lipoproteins which are increased in the postprandial hyperlipidemia. However, it has been shown over
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Analytical and bioanalytical chemistry, 406(25), 6203-6211 (2014-08-12)
High-performance affinity chromatography (HPAC) was utilized to examine the binding of very low density lipoprotein (VLDL) with drugs, using R/S-propranolol as a model. These studies indicated that two mechanisms existed for the binding of R- and S-propranolol with VLDL. The
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Journal of lipids, 2011, 189876-189876 (2011-07-21)
The heart is a major consumer of energy and is able to utilise a wide range of substrates including lipids. Nonesterified fatty acids (NEFA) were thought to be a favoured carbon source, but their quantitative contribution is limited because of
M C Sampedro et al.
Biochemical and biophysical research communications, 285(2), 393-399 (2001-07-11)
Human very-low-density lipoprotein (VLDL) inhibits DNA synthesis in lymphocytes activated by the nonspecific mitogen concanavalin A (Con A). We studied the effects of VLDL on lymphocyte activation (IL-2 receptor expression), cell cycle progression, and production of IL-2 and of IL-4
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