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Merck

RAB0556

Sigma-Aldrich

Human VWF / von Willebrand Factor ELISA Kit

for serum, plasma, cell culture supernatants and urine

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About This Item

UNSPSC-Code:
41116158
NACRES:
NA.32

Speziesreaktivität

human

Verpackung

kit of 96 wells (12 strips x 8 wells)

Methode(n)

ELISA: suitable

Aufnahme

sample type serum
sample type plasma
sample type cell culture supernatant(s)
sample type urine

assay range

inter-assay cv: <10%
intra-assay cv: <12%
sensitivity: 0.4 ng/mL

Nachweisverfahren

colorimetric

Versandbedingung

wet ice

Lagertemp.

−20°C

Angaben zum Gen

human ... VWF(7450)

Allgemeine Beschreibung

Human VWF/von Willebrand factor ELISA (Enzyme-Linked Immunosorbent Assay) kit is an in vitro assay for the quantitative measurement of a target protein in biological samples, such as serum, plasma, cell culture supernatants, urine and cell and tissue lysates.

Anwendung

For research use only. Not for use in diagnostic procedures.
Please refer to the attached General ELISA KIT Procedure (sandwich, competitive & Indirect ELISA)
Human VWF/von Willebrand factor ELISA kit has been used for the quantification of endothelial marker-von Willebrand factor in bone marrow-derived mesenchymal stem cells (BMSC) and human umbilical vein endothelial cells (HUVEC).

Sonstige Hinweise

A sample Certificate of Analysis is available for this product.
Please type the word sample in the text box provided for lot number.

Kit-Komponenten auch einzeln erhältlich

Produkt-Nr.
Beschreibung
SDB

  • RABTMB3ELISA Colorimetric TMB Reagent (HRP Substrate, Item H)SDB

  • RABSTOP3ELISA Stop Solution (Item I)SDB

  • RABELADAELISA 1X Assay/Sample Diluent Buffer A (Item D1)SDB

  • RABELADBELISA 5X Assay/Sample Diluent Buffer B (Item E1)SDB

  • RABWASH420X Wash Buffer (Item B)SDB

Piktogramme

Corrosion

Signalwort

Warning

H-Sätze

P-Sätze

Gefahreneinstufungen

Met. Corr. 1

Lagerklassenschlüssel

8A - Combustible, corrosive hazardous materials

WGK

WGK 3

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable


Analysenzertifikate (COA)

Suchen Sie nach Analysenzertifikate (COA), indem Sie die Lot-/Chargennummer des Produkts eingeben. Lot- und Chargennummern sind auf dem Produktetikett hinter den Wörtern ‘Lot’ oder ‘Batch’ (Lot oder Charge) zu finden.

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In der Dokumentenbibliothek finden Sie die Dokumentation zu den Produkten, die Sie kürzlich erworben haben.

Die Dokumentenbibliothek aufrufen

J W R Sins et al.
Journal of thrombosis and haemostasis : JTH, 15(7), 1392-1402 (2017-05-01)
Essentials The role of von Willebrand Factor (VWF) in the pathophysiology of sickle cell disease is unclear. We assessed markers of VWF during admission for vaso-occlusive crisis (VOC) and steady state. VWF reactivity was higher during VOC and was associated
Claus Rieker et al.
Arteriosclerosis, thrombosis, and vascular biology, 39(9), e195-e207 (2019-07-19)
The ApoE (apolipoprotein) allele epsilon 4 is a major genetic risk factor for Alzheimer disease, cardiovascular disorders, and stroke, indicating that it significantly impacts cerebral and vascular systems. However, very little is known about how APOE genotype affects brain endothelial
Erika Kajdácsi et al.
The Journal of allergy and clinical immunology, 133(6), 1686-1691 (2014-02-14)
Hereditary angioedema (HAE) caused by C1-inhibitor (C1-INH) deficiency (HAE-C1-INH) is a potentially life-threatening rare disease caused by the decreased activity of C1-INH. Lack of C1-INH leads to overproduction of bradykinin, a potent vasoactive peptide. Although angioedema is induced by bradykinin
Camila M de Andrade et al.
International journal of biological macromolecules, 134, 653-659 (2019-05-10)
Endothelium plays an important modulatory role due to the synthesis and secretion of molecules that act on hemostasis and fibrinolysis. As there are no literature data about the effect of crotoxin (CTX) - the main component of Crotalus durissus terrificus
Junchao Duan et al.
Nanotoxicology, 12(5), 470-484 (2018-04-17)
Nowadays, nanotechnology environmental health and safety (nanoEHS) is gaining attention. We previously found that silica nanoparticles (SiNPs) could induce vascular endothelial damage. However, the subsequent toxicologic response to SiNPs-induced endothelial damage was still largely unknown. In this study, we explored

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