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A5879

Sigma-Aldrich

2-(2-Aminoethyl)isothioharnstoff -dihydrobromid

Synonym(e):

2-(2-Aminoethyl)-2-thiopseudoharnstoff -dihydrobromid, AET, S-(2-Aminoethyl)-isothiouronium-bromid -hydrobromid

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About This Item

Lineare Formel:
NH2CH2CH2SC(=NH)NH2 · 2HBr
CAS-Nummer:
56-10-0
Molekulargewicht:
281.01
Beilstein:
3911163
EG-Nummer:
200-257-0
MDL-Nummer:
MFCD00037011
UNSPSC-Code:
12352200
PubChem Substanz-ID:
24278229
NACRES:
NA.77

Assay

≥98% (TLC)

Qualitätsniveau

100

Form

powder

mp (Schmelzpunkt)

190-196 °C (lit.)

Löslichkeit

water: 50 mg/mL, clear to very slightly hazy, colorless to yellow

SMILES String

Br[H].Br[H].NCCSC(N)=N

InChI

1S/C3H9N3S.2BrH/c4-1-2-7-3(5)6;;/h1-2,4H2,(H3,5,6);2*1H

InChIKey

XDVMCVGTDUKDHL-UHFFFAOYSA-N

Angaben zum Gen

human ... NOS1(4842) , NOS2(4843) , NOS2B(201288) , NOS2C(645740) , NOS3(4846)

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Anwendung

2-(2-Aminoethyl)isothiourea dihydrobromide inhibits nitric oxide synthase (NOS).

Biochem./physiol. Wirkung

Inhibits constitutive and inducible NOS.

Piktogramme

Exclamation mark
GHS07

Signalwort

Warning

Gefahreneinstufungen

Acute Tox. 4 Oral - Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3

Zielorgane

Respiratory system

Lagerklassenschlüssel

11 - Combustible Solids

WGK

WGK 3

Flammpunkt (°F)

Not applicable

Flammpunkt (°C)

Not applicable

Persönliche Schutzausrüstung

dust mask type N95 (US), Eyeshields, Gloves

Analysenzertifikate (COA)

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Sigma-Aldrich

H0262

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Hydroxyharnstoff 98%, powder

Sigma-Aldrich

H8627

Hydroxyharnstoff

O A Strand et al.
Scandinavian journal of clinical and laboratory investigation, 57(2), 105-109 (1997-04-01)
The effect of inhibiting nitric oxide (NO) synthesis during sepsis was studied in a caecal perforation model on Wistar rats. This model induces severe abdominal sepsis with a 48-h mortality > 90% in untreated animals. The survival time in hours
J L Fuentes et al.
Mutation research, 422(2), 339-345 (1998-12-05)
The effect of sodium diethyldithiocarbamate (DDC) and S-2-aminoethyl-isothiouronicadenosin-5-triphosphate (adeturon) in the induction of Escherichia coli SOS response promoted by gamma-irradiation was studied by measuring the induction of sulA gene and the induction of lambda prophage. Furthermore, as a way of
Y Gundersen et al.
The European journal of surgery = Acta chirurgica, 165(12), 1167-1174 (2000-01-15)
To examine the effects of the inducible nitric oxide synthase inhibitor aminoethyl-isothiourea (AE-ITU) on haemodynamic measurements, and correlate these with hepatic morphology and function in a porcine model of endotoxaemia. Experimental study. 15 juvenile pigs. Flow probes were placed around
C O Corso et al.
Journal of hepatology, 28(1), 61-69 (1998-04-16)
The question whether nitric oxide protects or impairs organ perfusion during early endotoxemia has not been completely answered. To evaluate the regulative function of nitric oxide on organ microvascular perfusion and leukocyte accumulation during endotoxemia, we studied the influence of
A Southey et al.
International journal of immunopharmacology, 19(11-12), 669-676 (1998-07-21)
Overproduction of nitric oxide (NO) by inducible nitric oxide synthase (iNOS) may contribute to the pathophysiology of ulcerative colitis. A 2,4,6-trinitrobenzenesulfonic acid sodium salt (TNBS) colitis model was established to examine the effect of selective iNOS inhibition, by S-(2-aminoethyl) isothiouronium
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