推薦產品
等級
reagent grade
產品線
Vetec™
化驗
98%
bp
135 °C (lit.)
mp
60-63 °C (lit.)
密度
0.901 g/mL at 25 °C (lit.)
SMILES 字串
C\C(C)=N/O
InChI
1S/C3H7NO/c1-3(2)4-5/h5H,1-2H3
InChI 密鑰
PXAJQJMDEXJWFB-UHFFFAOYSA-N
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法律資訊
Vetec is a trademark of Merck KGaA, Darmstadt, Germany
訊號詞
Danger
危險分類
Acute Tox. 4 Dermal - Carc. 2 - Eye Dam. 1 - Flam. Sol. 2 - Skin Sens. 1B
儲存類別代碼
4.1B - Flammable solid hazardous materials
水污染物質分類（WGK）
WGK 3
閃點（°F）
Not applicable
閃點（°C）
Not applicable
Stefanie Zorbas-Seifried et al.
Molecular pharmacology, 71(1), 357-365 (2006-10-20)
The presence of cis-configured exchangeable ligands has long been considered a prerequisite for antitumor activity of platinum complexes, but over the past few years, several examples violating this structure-activity relationship have been recognized. We report here on studies with the
C Kohl et al.
Carcinogenesis, 13(7), 1091-1094 (1992-07-01)
The hepatocarcinogenicity of acetoxime has been tentatively linked with its metabolic oxidation to the potent genotoxicant and carcinogen propane 2-nitronate (P2-N). In order to test the hypothesis that acetoxime is metabolized to P2-N, the oxime (20 mM) was incubated with
R S Sodum et al.
Chemical research in toxicology, 10(12), 1420-1426 (1998-01-23)
Previously, the secondary nitroalkane 2-nitropropane, a strong hepatocarcinogen in rats, had been shown to induce the formation of 8-aminoguanine in both DNA and RNA of rat liver through a sulfotransferase-mediated pathway. This pathway was postulated to convert the carcinogen into
S S Mirvish et al.
Journal of the National Cancer Institute, 69(4), 961-962 (1982-10-01)
Acetoxime was tested for carcinogenicity by chronic administration in the drinking water to male and female outbred MRC-Wistar rats. The dose of 1.0 g/liter was administered 5 days/week for 18 months (total dose, 6.2--7.0 g/rat). The test compound induced benign
P Kreis et al.
Carcinogenesis, 21(2), 295-299 (2000-02-05)
The industrial solvent 2-nitropropane (2-NP) is a genotoxic hepatocarcinogen in rats. The genotoxicity of the compound in rats has been attributed to sulfotransferase-mediated formation of DNA-reactive nitrenium ions from the anionic form of 2-NP, propane 2-nitronate (P2N). Whether human sulfotransferases
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