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Key Documents

F0257

Sigma-Aldrich

抗小鼠IgG(全分子)–FITC 山羊抗

affinity isolated antibody, buffered aqueous solution

同義詞:

山羊中产生的抗小鼠FITC-抗小鼠IgG(全分子)-FITC抗体, 抗小鼠FITC

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About This Item

MDL號碼:
分類程式碼代碼:
12352203
NACRES:
NA.46

生物源

goat

共軛

FITC conjugate

抗體表格

affinity isolated antibody

抗體產品種類

secondary antibodies

無性繁殖

polyclonal

形狀

buffered aqueous solution

技術

direct immunofluorescence: 1:32

儲存溫度

2-8°C

目標翻譯後修改

unmodified

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一般說明

IgG抗体亚型是免疫系统中含量最高的血清免疫球蛋白。它由B细胞分泌,存在于血液和细胞外液中,可防止细菌、真菌和病毒引起的感染。母体IgG可通过胎盘转移至胎儿,胎盘对新生儿免疫防御感染至关重要。小鼠IgG具有四种不同的同种型,即IgG1、IgG2a、IgG2b和IgG3。

免疫原

纯化的小鼠IgG

應用

抗小鼠IgG(全分子)-FITC抗体可用于:
  • 免疫组织化学法(1:200)(1:500)
  • 免疫荧光法(1:100)(1:500)(1:200)
  • 免疫细胞化学法(1:1000)

外觀

溶于含有1%牛血清白蛋白和15mM叠氮化钠的0.01M磷酸盐缓冲盐水(pH7.4)中。

免責聲明

除非我们的产品目录或产品随附的其他公司文档中另有说明,否则我们的产品仅供研究使用,不得用于任何其他目的,包括但不限于未经授权的商业用途、体外诊断用途、离体或体内治疗用途或任何类型的消费或应用于人或动物。

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儲存類別代碼

10 - Combustible liquids

水污染物質分類(WGK)

WGK 2

閃點(°F)

Not applicable

閃點(°C)

Not applicable


分析證明 (COA)

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存取文件庫

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Specific heat shock proteins protect microtubules during simulated ischemia in cardiac myocytes
Bluhm, W., F., et al.
American Journal of Physiology. Heart and Circulatory Physiology, 275(6), H2243-H2249 (1998)
Susana M Garcia et al.
Genes & development, 21(22), 3006-3016 (2007-11-17)
Protein homeostasis maintains proper intracellular balance by promoting protein folding and clearance mechanisms while minimizing the stress caused by the accumulation of misfolded and damaged proteins. Chronic expression of aggregation-prone proteins is deleterious to the cell and has been linked
Maruthachalam Ravi et al.
PLoS genetics, 7(6), e1002121-e1002121 (2011-06-23)
Centromere behavior is specialized in meiosis I, so that sister chromatids of homologous chromosomes are pulled toward the same side of the spindle (through kinetochore mono-orientation) and chromosome number is reduced. Factors required for mono-orientation have been identified in yeast.
Siu-Kei Chow et al.
ASN neuro, 2(1), e00026-e00026 (2009-12-17)
The contribution of astrocytes to the pathophysiology of AD (Alzheimer's disease) and the molecular and signalling mechanisms that potentially underlie them are still very poorly understood. However, there is mounting evidence that calcium dysregulation in astrocytes may be playing a

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