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C7057

Nunc® Lab-Tek® II\腔室载玻片系统

wells 8, well area 0.7 cm2, sterile

同義詞:

显微镜载玻片, 细胞培养腔, 腔室载玻片

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About This Item

分類程式碼代碼:
41161502
NACRES:
NB.22

材料

polystyrene chamber

無菌

sterile

特點

CE marked

製造商/商標名

Nunc 154534

包裝

pack of 16 ×

孔面積

0.7 cm2

8

有效容積

0.2-0.5 mL

適合性

suitable for (cell culture; attachment and growth of cells)

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一般說明

Nunc® Lab-Tek® II腔室载玻片系统

  • 可拆除的聚苯乙烯培养基腔室,有1、2、4和8孔四种规格
  • 无荧光的显微镜载玻片,为圆角玻璃(25×75×1.2 mm)
  • 生物相容性粘合剂
  • 聚苯乙烯封盖
  • 在玻片上印有惰性的疏水孔隔板
  • Superfrost® 印刷书写区域
  • 经过处理,适合细胞的附着和生长
  • 每个包装中配有载玻片分离器

法律資訊

Chamber Slide is a trademark of Thermo Fisher Scientific or its subsidiaries
Lab-Tek is a registered trademark of Thermo Fisher Scientific or its subsidiaries
Nunc is a registered trademark of Thermo Fisher Scientific or its subsidiaries
Superfrost is a registered trademark of Erie Scientific Co.

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Angelo G Torrente et al.
The Journal of physiology, 595(12), 3847-3865 (2017-03-28)
Repolarizing currents through K+ channels are essential for proper sinoatrial node (SAN) pacemaking, but the influence of intracellular Ca2+ on repolarization in the SAN is uncertain. We identified all three isoforms of Ca2+ -activated small conductance K+ (SK) channels in
L S Mamsen et al.
Molecular human reproduction, 21(7), 571-582 (2015-04-30)
From early embryonic life, anti-Müllerian hormone (AMH) is produced by Sertoli cells and is essential for male sex differentiation. In females, AMH is produced by immature granulosa cells (GCs) but a definitive function in females is uncertain. We have assessed
Philip E D Chung et al.
Nature communications, 11(1), 1825-1825 (2020-04-15)
Pineoblastoma is a rare pediatric cancer induced by germline mutations in the tumor suppressors RB1 or DICER1. Presence of leptomeningeal metastases is indicative of poor prognosis. Here we report that inactivation of Rb plus p53 via a WAP-Cre transgene, commonly
Sebastian Bäumer et al.
Clinical cancer research : an official journal of the American Association for Cancer Research, 21(6), 1383-1394 (2015-01-16)
KRAS mutations are frequent driver mutations in multiple cancers. KRAS mutations also induce anti-EGFR antibody resistance in adenocarcinoma such as colon cancer. The aim of this study was to overcome anti-EGFR antibody resistance by coupling the antibody to KRAS-specific siRNA.

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