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10108294001

Roche

磷酸烯醇丙酮酸

97% (PEP-K), pkg of 1 g

同義詞:

磷烯醇丙酮酸 单钾盐, 2-(膦酰氧基)-2-丙烯酸 单钾盐, 磷酸烯醇丙酮酸单钾盐, PEP-K

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About This Item

經驗公式(希爾表示法):
C3H4KO6P
CAS號碼:
分子量::
206.13
Beilstein:
4603446
MDL號碼:
分類程式碼代碼:
12352204
PubChem物質ID:

描述

C3H4O6PK (Formula)
PEP, monopotassium salt

品質等級

化驗

97% (PEP-K)

形狀

solid

分子量

Mr 168.0 (PEP)
Mr 206.1 (PEP-K)

包裝

pkg of 1 g

製造商/商標名

Roche

儲存溫度

2-8°C

SMILES 字串

[K+].OC(=O)C(=C)OP(O)([O-])=O

InChI

1S/C3H5O6P.K/c1-2(3(4)5)9-10(6,7)8;/h1H2,(H,4,5)(H2,6,7,8);/q;+1/p-1

InChI 密鑰

SOSDSEAIODNVPX-UHFFFAOYSA-M

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一般說明

内容物: 80%PEP(酶法)

應用

丙酮酸激酶的底物。
磷酸烯醇式丙酮酸 (PEP) 已用于:
  • 谷氨酸-半胱氨酸连接酶活性
  • v-ATPase 酶活性测定
  • meso 激酶测定

生化/生理作用

磷酸烯醇丙酮酸 (PEP) 在代谢中起重要作用。它具有较高的磷酸基团转移潜能,在C-C,C-P 和 C-O 键形成反应中起着通用的 C3-合成子的作用。PEP 在糖酵解的最后一步充当丙酮酸激酶 (PK) 的底物。

其他說明

仅用于生命科学研究。不可用于诊断。

儲存類別代碼

11 - Combustible Solids

水污染物質分類(WGK)

WGK 1

閃點(°F)

Not applicable

閃點(°C)

Not applicable


分析證明 (COA)

輸入產品批次/批號來搜索 分析證明 (COA)。在產品’s標籤上找到批次和批號,寫有 ‘Lot’或‘Batch’.。

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存取文件庫

Synthesis of phosphoenol pyruvate (PEP) analogues and evaluation as inhibitors of PEP-utilizing enzymes.
Garcia A, et al.
FEBS Journal, 269(13), 3226-3236 (2002)
Glutathione participates in the modulation of starvation-induced autophagy in carcinoma cells.
Desideri E
Autophagy, 8(12), 1769-1781 (2012)
Maria B Bagh et al.
Nature communications, 8, 14612-14612 (2017-03-08)
Defective lysosomal acidification contributes to virtually all lysosomal storage disorders (LSDs) and to common neurodegenerative diseases like Alzheimer's and Parkinson's. Despite its fundamental importance, the mechanism(s) underlying this defect remains unclear. The v-ATPase, a multisubunit protein complex composed of cytosolic
Jasmine M Hershewe et al.
Nature communications, 12(1), 2363-2363 (2021-04-24)
Cell-free gene expression (CFE) systems from crude cellular extracts have attracted much attention for biomanufacturing and synthetic biology. However, activating membrane-dependent functionality of cell-derived vesicles in bacterial CFE systems has been limited. Here, we address this limitation by characterizing native
Trace Thome et al.
American journal of physiology. Cell physiology, 317(4), C701-C713 (2019-07-11)
Chronic kidney disease (CKD) leads to increased skeletal muscle fatigue, weakness, and atrophy. Previous work has implicated mitochondria within the skeletal muscle as a mediator of muscle dysfunction in CKD; however, the mechanisms underlying mitochondrial dysfunction in CKD are not

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