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重要文件

AB9328

Sigma-Aldrich

Anti-Thioredoxin 1 Antibody

serum, Chemicon®

同義詞:

TRX1

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About This Item

分類程式碼代碼:
12352203
eCl@ss:
32160702
NACRES:
NA.41
無性繁殖:
polyclonal
application:
WB
物種活性:
mouse, rat
技術:
western blot: suitable
citations:
2

生物源

rabbit

品質等級

抗體表格

serum

抗體產品種類

primary antibodies

無性繁殖

polyclonal

物種活性

mouse, rat

物種活性(以同源性預測)

human (sequence homology 14/15)

製造商/商標名

Chemicon®

技術

western blot: suitable

UniProt登錄號

運輸包裝

dry ice

目標翻譯後修改

unmodified

基因資訊

human ... TXN(7295)

特異性

Recognizes Thioredoxin 1 (TRX1).

免疫原

Synthetic peptide from rat/mouse Thioredoxin 1.

應用

Detect Thioredoxin 1 using this Anti-Thioredoxin 1 Antibody validated for use in WB.
Immunoblotting: 1:1,000-1:5,000 using ECL. Reacts with the ~12-14 kD thioredoxin 1 protein. Additional bands >30 kD may been seen.

Suggested dilution and blocking buffer is TBST containing 5% non-fat milk. Suggested gel percentage is 15%. Suggested incubation time is overnight.
Research Category
Neuroscience
Research Sub Category
Oxidative Stress

聯結

Replaces: MABN1090

外觀

Rabbit serum. Liquid. Contains no preservative.

儲存和穩定性

Maintain at -20°C in undiluted aliquots for up to 6 months after date of receipt. Avoid repeated freeze/thaw cycles.

法律資訊

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

免責聲明

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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儲存類別代碼

10 - Combustible liquids

水污染物質分類(WGK)

WGK 1

閃點(°F)

Not applicable

閃點(°C)

Not applicable


分析證明 (COA)

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Chiara Milanese et al.
Nature communications, 10(1), 4887-4887 (2019-10-28)
Accumulation of DNA lesions causing transcription stress is associated with natural and accelerated aging and culminates with profound metabolic alterations. Our understanding of the mechanisms governing metabolic redesign upon genomic instability, however, is highly rudimentary. Using Ercc1-defective mice and Xpg

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