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Key Documents

609358

Sigma-Aldrich

氰酸钾-15N

≥98 atom % 15N, ≥95% (CP)

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About This Item

線性公式:
KOC15N
CAS號碼:
分子量::
82.11
MDL號碼:
分類程式碼代碼:
12352118
PubChem物質ID:

同位素純度

≥98 atom % 15N

化驗

≥95% (CP)

形狀

solid

質量偏移

M+1

SMILES 字串

[K+].[O-]C#[15N]

InChI

1S/CHNO.K/c2-1-3;/h3H;/q;+1/p-1/i2+1;

InChI 密鑰

GKKCIDNWFBPDBW-CGOMOMTCSA-M

包裝

This product may be available from bulk stock and can be packaged on demand. For information on pricing, availability and packaging, please contact Stable Isotopes Customer Service.

象形圖

Exclamation mark

訊號詞

Warning

危險聲明

危險分類

Acute Tox. 4 Oral

儲存類別代碼

11 - Combustible Solids

水污染物質分類(WGK)

WGK 3

閃點(°F)

Not applicable

閃點(°C)

Not applicable

個人防護裝備

dust mask type N95 (US), Eyeshields, Gloves


分析證明 (COA)

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F G Schimitel et al.
Neuroscience, 200, 59-73 (2011-11-09)
Dyspnea, hunger for air, and urge to flee are the cardinal symptoms of panic attacks. Patients also show baseline respiratory abnormalities and a higher rate of comorbid and antecedent respiratory diseases. Panic attacks are also precipitated by infusion of sodium
LAIS Allergoid.
G Mistrello
International journal of immunopathology and pharmacology, 18(4 Suppl), 21-24 (2007-12-06)
Małgorzata Iciek et al.
Human & experimental toxicology, 30(10), 1601-1608 (2010-12-24)
Cyanate and its active form isocyanate are formed mainly in the process of nonenzymatic urea biodegradation. Cyanate is capable of protein S- and N-carbamoylation, which can affect their activity. The present studies aimed to demonstrate the effect of cyanate on
Josiane C Cruz et al.
Autonomic neuroscience : basic & clinical, 146(1-2), 47-55 (2009-01-10)
GABAergic, nitrergic and glutamatergic mechanisms in the PVN on the baseline mean arterial pressure (MAP), heart rate (HR) and on the cardiovascular responses to chemoreflex activation in awake rat were evaluated. Chemoreflex was activated with KCN before and after microinjections
H L Liang et al.
Neuroscience, 153(4), 963-974 (2008-04-29)
Parkinson's disease is a common progressive neurodegenerative disorder characterized by the degeneration of dopaminergic neurons in the substantia nigra pars compacta. Mitochondrial dysfunction has been strongly implicated in the pathogenesis of Parkinson's disease. Thus, therapeutic approaches that improve mitochondrial function

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