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重要文件

244589

Sigma-Aldrich

氯化锰

powder and chunks, ≥99% trace metals basis

同義詞:

二氯化锰, 黄钙橄石

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About This Item

線性公式:
MnCl2
CAS號碼:
分子量::
125.84
MDL號碼:
分類程式碼代碼:
12352302
eCl@ss:
38190105
PubChem物質ID:
NACRES:
NA.23

等級

for analytical purposes

化驗

≥99% trace metals basis

形狀

powder and chunks

雜質

≤10000.0 ppm Trace Metal Analysis

mp

652 °C (lit.)

密度

2.98 g/mL at 25 °C (lit.)

應用

battery manufacturing

SMILES 字串

Cl[Mn]Cl

InChI

1S/2ClH.Mn/h2*1H;/q;;+2/p-2

InChI 密鑰

GLFNIEUTAYBVOC-UHFFFAOYSA-L

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一般說明

氯化锰(II)是一种粉红色结晶固体,极易溶于水。无水形式的氯化锰(II)具有与层状氯化镉类似的结构。它是合成有机锰化合物的关键原料。

應用

氯化锰(II)可用于:
  • 作为合成氧化锰 纳米颗粒的前体。
  • 制备用于超级电容器的Mn(II)配位聚苯胺 电极。
  • 制备用于锂离子电池的热激活高阳极。 性能MOF
  • 作为合成光学性能增强的聚合物复合膜的原料。

訊號詞

Danger

危險聲明

危險分類

Acute Tox. 3 Oral - Eye Dam. 1 - STOT RE 2

標靶器官

Brain

儲存類別代碼

6.1D - Non-combustible acute toxic Cat.3 / toxic hazardous materials or hazardous materials causing chronic effects

水污染物質分類(WGK)

WGK 2

閃點(°F)

Not applicable

閃點(°C)

Not applicable

個人防護裝備

dust mask type N95 (US), Eyeshields, Faceshields, Gloves


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分析證明 (COA)

Lot/Batch Number

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Edward Pajarillo et al.
Neurotoxicology, 65, 280-288 (2017-12-01)
Chronic exposure to manganese (Mn) causes neurotoxicity, referred to as manganism, with common clinical features of parkinsonism. 17β-estradiol (E2) and tamoxifen (TX), a selective estrogen receptor modulator (SERM), afford neuroprotection in several neurological disorders, including Parkinson's disease (PD). In the
Abbati, G.L. et al.
Inorganic Chemistry, 37, 1430-1430 (1998)
Pao-Chun Lin et al.
mAbs, 11(5), 965-976 (2019-05-03)
Chinese hamster ovary (CHO) cells are the biopharmaceutical industry's primary means of manufacturing therapeutic proteins, including monoclonal antibodies. The major challenge in cell line development for the production of recombinant biopharmaceuticals lies in generating and isolating rare high-producing stable clones
Tang, J. et al.
Tetrahedron, 55, 1893-1893 (1999)
Marta Sidoryk-Wegrzynowicz et al.
Journal of neurochemistry, 122(4), 856-867 (2012-06-20)
Manganese (Mn) has been implicated in the impairment of the glutamate-glutamine cycling (GGC) by deregulation of Glu and glutamine (Gln) turnover in astrocytes. Here, we have examined possible mechanisms involved in the Mn(II)-mediated disruption of Glu turnover, including those related

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