Skip to Content
Merck
All Photos(3)

Documents

A21207

Sigma-Aldrich

3-Acetylpyridine

98%

Synonym(s):

Methyl 3-pyridyl ketone

Sign Into View Organizational & Contract Pricing
All Photos(3)

About This Item

Empirical Formula (Hill Notation):
C7H7NO
CAS Number:
350-03-8
Molecular Weight:
121.14
Beilstein:
107751
EC Number:
206-496-7
MDL number:
MFCD00006396
UNSPSC Code:
12352100
PubChem Substance ID:
24890625
NACRES:
NA.22

Assay

98%

form

liquid

refractive index

n20/D 1.534 (lit.)

bp

220 °C (lit.)

mp

11-13 °C (lit.)

density

1.102 g/mL at 25 °C (lit.)

SMILES string

CC(=O)c1cccnc1

InChI

1S/C7H7NO/c1-6(9)7-3-2-4-8-5-7/h2-5H,1H3

InChI key

WEGYGNROSJDEIW-UHFFFAOYSA-N

Looking for similar products? Visit Product Comparison Guide

Related Categories

Pictograms

Skull and crossbones
GHS06

Signal Word

Danger

Hazard Statements

H301,H315

Hazard Classifications

Acute Tox. 3 Oral - Skin Irrit. 2

Storage Class Code

6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects

WGK

WGK 1

Flash Point(F)

219.2 °F - closed cup

Flash Point(C)

104 °C - closed cup

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

Already Own This Product?

Find documentation for the products that you have recently purchased in the Document Library.

Visit the Document Library

Matasha Dhar et al.
Nature communications, 7, 12938-12938 (2016-09-22)
Neurodegenerative lesions induce sprouting of new collaterals from surviving axons, but the extent to which this form of axonal remodelling alters brain functional structure remains unclear. To understand how collateral sprouting proceeds in the adult brain, we imaged post-lesion sprouting
Iran Goudarzi et al.
European journal of pharmacology, 642(1-3), 56-65 (2010-07-20)
Electrophysiological dysfunction of Purkinje cells causes cerebellar ataxia. Recent studies indicated that 4-aminopyridine (4-AP) can prevent the attacks in patients with episodic ataxia type 2. However, the cellular mechanism(s) by which 4-AP might be beneficial for the improvement of motor
Yi Zhang et al.
American journal of physiology. Heart and circulatory physiology, 285(3), H1177-H1182 (2003-05-17)
We sought to define the contribution of the climbing fibers (CF), one of the major inputs to Purkinje neurons, to the increase in cerebellar blood flow (BFcrb) produced by activation of the cerebellar cortex. The neurotoxin 3-acetylpyridine was used to
Brian H Hess et al.
Brain research, 986(1-2), 54-62 (2003-09-11)
The temporally protracted heredodegeneration of cerebellar Purkinje cells in shaker mutant rats can be modified: ablation of the inferior olive accelerates their degeneration whereas chronic intraventricular infusion of trophic factors extends their survival. The present study sought to determine if
M Kaffashian et al.
Physiological research, 60(2), 355-365 (2010-12-01)
Alterations in the intrinsic properties of Purkinje cells (PCs) may contribute to the abnormal motor performance observed in ataxic rats. To investigate whether such changes in the intrinsic neuronal excitability could be attributed to the role of Ca(2+)-activated K(+) channels
View All Related Papers

Our team of scientists has experience in all areas of research including Life Science, Material Science, Chemical Synthesis, Chromatography, Analytical and many others.

Contact Technical Service