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3951

Sigma-Aldrich

Periodic Acid Solution

1 g/dL in deionized water

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About This Item

MDL number:
MFCD00011344
UNSPSC Code:
12352106
PubChem Substance ID:
24864643
NACRES:
NA.47

form

solution

Quality Level

500

shelf life

expiry date on label

reaction suitability

reagent type: oxidant

IVD

for in vitro diagnostic use

concentration

1 g/dL in deionized water

pH

1.77

application(s)

hematology
histology

shipped in

wet ice

storage temp.

2-8°C

SMILES string

OI(O)(O)(O)(O)=O

InChI

1S/H5IO6/c2-1(3,4,5,6)7/h(H5,2,3,4,5,6,7)

InChI key

TWLXDPFBEPBAQB-UHFFFAOYSA-N

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Application

For use in procedure no. 395 for Periodic Acid-Schiff (PAS) and Silver Stain Procedure No. HT100.

Pictograms

Corrosion
GHS05

Signal Word

Danger

Hazard Statements

H314 - H412

Hazard Classifications

Aquatic Chronic 3 - Eye Dam. 1 - Skin Corr. 1

Storage Class Code

8B - Non-combustible, corrosive hazardous materials

WGK

WGK 2

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Globoid cell leukodystrophy (GLD), or Krabbe disease, is an inherited, neurologic disorder that results from deficiency of a lysosomal enzyme, galactosylceramidase. Most commonly, deficits of galactosylceramidase result in widespread central and peripheral nervous system demyelination and death in affected infants
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Globoid cell leukodystrophy (GLD), or Krabbe's disease, is a debilitating and always fatal pediatric neurodegenerative disease caused by a mutation in the gene encoding the hydrolytic enzyme galactosylceramidase (GALC). In the absence of GALC, progressive loss of myelin and accumulation
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Skeletal muscle is a dynamic tissue the size of which can be remodeled through the concerted actions of various cues. Here, we investigated the skeletal muscle transcriptional program and identified key tissue-specific regulatory genetic elements. Our results show that Myod1
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