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等級
ACS reagent
anhydrous
品質等級
描述
anionic
產品線
Redi-Dri™
化驗
≥99.0%
品質
free-flowing
分子量
288.38 g/mol
雜質
≤0.06 meq/g Titr. Base
≤4.0% unsulfated alcohols
≥96.0% fatty alcohols
損耗
≤1.0% loss on drying
mp
204-207 °C (lit.)
溶解度
water: soluble 100 mg/mL (opalescent solution)
正離子痕跡
heavy metals (as Pb): ≤0.002%
&lambda ;
3 %H2O reference
紫外吸收
λ: 230-350 nm Amax: ≤0.1
SMILES 字串
[Na+].CCCCCCCCCCCCOS([O-])(=O)=O
InChI
1S/C12H26O4S.Na/c1-2-3-4-5-6-7-8-9-10-11-12-16-17(13,14)15;/h2-12H2,1H3,(H,13,14,15);/q;+1/p-1
InChI 密鑰
DBMJMQXJHONAFJ-UHFFFAOYSA-M
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一般說明
SDS(十二烷基硫酸钠)是一种润湿剂和一种阴离子洗涤剂,在碱性和酸性溶液中均能有效发挥作用。它通常用于脂类和蛋白质的增溶。它也可以作为有效的蛋白质变性剂。SDS, volume-wise 是最常用的合成表面活性剂之一。
應用
阴离子洗涤剂
法律資訊
Redi-Dri is a trademark of Sigma-Aldrich Co. LLC
訊號詞
Danger
危險分類
Acute Tox. 4 Oral - Aquatic Chronic 3 - Eye Dam. 1 - Skin Irrit. 2
儲存類別代碼
11 - Combustible Solids
水污染物質分類(WGK)
WGK 2
閃點(°F)
338.0 °F
閃點(°C)
170 °C
其他客户在看
Toxicity of anionic detergents determined by Saccharomyces cerevisiae microarray analysis.
Water Research, 38(1), 61-70 (2004)
Journal of hazardous materials, 370, 42-53 (2018-09-15)
This study investigates Bisphenol A (BPA) induced oxidative stress that mediates the genotoxicity in in vivo model Drosophila melanogaster. The calculated LC50 for BPA was 12.35 μg/mL. The strains of D. melanogaster were reared in 0.1, 1.0, 2.5 and 5.0 μg/mL BPA
Biochimica et biophysica acta, 733(2), 210-215 (1983-09-07)
Over 50 detergents were tested to establish which would be most effective in releasing proteins from membrane-bounded compartments without denaturing them. Various concentrations each of detergent were tested for two activities: (1) solubilization of egg phospholipid liposomes as measured by
Cell reports, 25(2), 328-338 (2018-10-12)
Cancer cell migration through and away from tumors is driven in part by migration along aligned extracellular matrix, a process known as contact guidance (CG). To concurrently study the influence of architectural and mechanical regulators of CG sensing, we developed
Cancer cell, 34(1), 69-84 (2018-07-17)
Human melanomas frequently harbor amplifications of EZH2. However, the contribution of EZH2 to melanoma formation has remained elusive. Taking advantage of murine melanoma models, we show that EZH2 drives tumorigenesis from benign BrafV600E- or NrasQ61K-expressing melanocytes by silencing of genes
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