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等級
pharmaceutical primary standard
API 家族
modafinil
製造商/商標名
EDQM
應用
pharmaceutical (small molecule)
格式
neat
儲存溫度
2-8°C
SMILES 字串
NC(=O)CS(=O)C(c1ccccc1)c2ccccc2
InChI
1S/C15H15NO2S/c16-14(17)11-19(18)15(12-7-3-1-4-8-12)13-9-5-2-6-10-13/h1-10,15H,11H2,(H2,16,17)
InChI 密鑰
YFGHCGITMMYXAQ-UHFFFAOYSA-N
基因資訊
human ... SLC6A3(6531)
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一般說明
This product is provided as delivered and specified by the issuing Pharmacopoeia. All information provided in support of this product, including SDS and any product information leaflets have been developed and issued under the Authority of the Issuing Pharmacopoeia. For further information and support please go to the website of the issuing Pharmacopoeia.
應用
Modafinil for system suitability EP Reference standard, intended for use in laboratory tests only as specifically prescribed in the European Pharmacopoeia.
生化/生理作用
Modafinil is a central nervous system vigilance promoting agent, which posseses neuroprotective properties.
包裝
The product is delivered as supplied by the issuing Pharmacopoeia. For the current unit quantity, please visit the EDQM reference substance catalogue.
其他說明
Sales restrictions may apply.
訊號詞
Warning
危險聲明
危險分類
Acute Tox. 4 Oral - Repr. 2
儲存類別代碼
11 - Combustible Solids
水污染物質分類(WGK)
WGK 3
閃點(°F)
Not applicable
閃點(°C)
Not applicable
British journal of clinical pharmacology, 75(3), 728-737 (2012-10-10)
Over recent years there has been increasing research into both pharmaceutical and nutraceutical cognition enhancers. Here we aimed to calculate the effect sizes of positive cognitive effect of the pharmaceutical modafinil in order to benchmark the effect of two widely
Cognitive enhancers are set to change workplaces.
BMJ (Clinical research ed.), 345, e7538-e7538 (2012-11-09)
Editorial: Cognitive enhancement: are we barking up the wrong tree?
Current drug abuse reviews, 5(4), 255-256 (2012-12-21)
Modafinil #259.
Journal of palliative medicine, 15(12), 1388-1389 (2012-12-05)
PloS one, 7(9), e45471-e45471 (2012-10-03)
We have previously shown that modafinil promotes wakefulness via dopamine receptor D(1) and D(2) receptors; however, the locus where dopamine acts has not been identified. We proposed that the nucleus accumbens (NAc) that receives the ventral tegmental area dopamine inputs
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