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Merck

BR701330

BRAND® 96 孔微孔板,U 形底

round bottom, non-sterile

别名:

微量板, 板子, 样品储存

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About This Item

分類程式碼代碼:
41121800
NACRES:
NB.15

材料

polypropylene
round bottom

無菌

non-sterile

包裝

pack of 100 ea

製造商/商標名

BRAND 701330

尺寸

96 well

孔最大容積

300 μm

結合類型

non-treated surface

相关类别

一般說明

PP. For volumes up to 300 µl. Compatible with virtually all leading microplate centrifuges. Raised rings around the orifice of each well minimize possible cross-contamination. The plates can be sealed using self-adhesive films, such as DMSO-resistant sealing film (cross-cut) with alphanumeric coding.

特點和優勢

  • BRAND® 96 孔微孔板,U 形底是一种深孔板,非常适合凝集和其他需要搅拌和洗涤样品的测定。
  • 具有圆形井底,没有边缘。
  • 孔口周围的凸起环可防止可能的交叉污染。
  • 与一系列微孔板离心机兼容。
  • 可使用合适的密封垫或自粘密封膜牢固密封。
  • 用于准确识别的字母数字代码。
  • 由高透明度的医用级聚丙烯(PP) 制成,有助于提高样品的可见性。
  • 耐大多数溶剂,包括 DMSO、苯酚和氯仿。

法律資訊

BRAND is a registered trademark of BRAND GMBH + CO KG

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Julien Racle et al.
Nature biotechnology, 37(11), 1283-1286 (2019-10-16)
Predictions of epitopes presented by class II human leukocyte antigen molecules (HLA-II) have limited accuracy, restricting vaccine and therapy design. Here we combined unbiased mass spectrometry with a motif deconvolution algorithm to profile and analyze a total of 99,265 unique
Timo Strohäker et al.
Nature communications, 10(1), 5535-5535 (2019-12-05)
Parkinson's disease (PD) and Multiple System Atrophy (MSA) are clinically distinctive diseases that feature a common neuropathological hallmark of aggregated α-synuclein. Little is known about how differences in α-synuclein aggregate structure affect disease phenotype. Here, we amplified α-synuclein aggregates from PD
Mateusz Maciejczyk et al.
Oxidative medicine and cellular longevity, 2019, 4393460-4393460 (2019-12-31)
Despite the proven role of oxidative stress in numerous systemic diseases and in the process of aging, little is still known about the salivary redox balance of healthy children, adults, and the elderly. Our study was the first to assess
Lisa-Marie Mauracher et al.
Journal of clinical medicine, 8(10) (2019-10-05)
The exact contribution of neutrophils to post-resuscitative brain damage is unknown. We aimed to investigate whether neutrophil extracellular trap (NET) formation in the early phase after return of spontaneous circulation (ROSC) may be associated with poor 30 day neurologic function
Gefei Chen et al.
Communications biology, 3(1), 32-32 (2020-01-22)
Molecular chaperones play important roles in preventing protein misfolding and its potentially harmful consequences. Deterioration of molecular chaperone systems upon ageing are thought to underlie age-related neurodegenerative diseases, and augmenting their activities could have therapeutic potential. The dementia relevant domain

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