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Merck

450995

Sigma-Aldrich

氯化锰

AnhydroBeads, −10 mesh, 99.999% trace metals basis

别名:

二氯化锰, 黄钙橄石

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About This Item

线性分子式:
MnCl2
CAS号:
分子量:
125.84
MDL號碼:
分類程式碼代碼:
12352302
PubChem物質ID:
NACRES:
NA.23

產品線

AnhydroBeads

化驗

99.999% trace metals basis

雜質

≤15.0 ppm Trace Metal Analysis

粒徑

−10 mesh

mp

652 °C (lit.)

密度

2.98 g/mL at 25 °C (lit.)

SMILES 字串

Cl[Mn]Cl

InChI

1S/2ClH.Mn/h2*1H;/q;;+2/p-2

InChI 密鑰

GLFNIEUTAYBVOC-UHFFFAOYSA-L

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應用

通过 MnCl2 氧化及同时进行的 NaOMe 促进型聚集来合成一种新型高自旋六核 Mn(III) 原子簇化合物 [NaMn6(OMe)12(dbm)6]+。该原子簇化合物具有全新的环状结构,在基态时具有 24 个不成对电子。还用于形成可用于自由基环化反应的活性 Mn(0) 物质。

法律資訊

AnhydroBeads is a trademark of Sigma-Aldrich Co. LLC

訊號詞

Danger

危險聲明

危險分類

Acute Tox. 3 Oral - Eye Dam. 1 - STOT RE 2

標靶器官

Brain

儲存類別代碼

6.1D - Non-combustible acute toxic Cat.3 / toxic hazardous materials or hazardous materials causing chronic effects

水污染物質分類(WGK)

WGK 2

閃點(°F)

Not applicable

閃點(°C)

Not applicable

個人防護裝備

dust mask type N95 (US), Eyeshields, Faceshields, Gloves


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Abbati, G.L. et al.
Inorganic Chemistry, 37, 1430-1430 (1998)
Tang, J. et al.
Tetrahedron, 55, 1893-1893 (1999)
Jerome A Roth et al.
Toxicology in vitro : an international journal published in association with BIBRA, 26(7), 1143-1149 (2012-07-31)
Mutations in the parkin gene are linked to development of juvenile onset of Parkinson's disease and recent studies have reported that parkin can protect against increased oxidative stress and mitochondrial dysfunction caused by a variety of oxidative and toxic insults.
Marta Sidoryk-Wegrzynowicz et al.
Journal of neurochemistry, 122(4), 856-867 (2012-06-20)
Manganese (Mn) has been implicated in the impairment of the glutamate-glutamine cycling (GGC) by deregulation of Glu and glutamine (Gln) turnover in astrocytes. Here, we have examined possible mechanisms involved in the Mn(II)-mediated disruption of Glu turnover, including those related
Jerome A Roth et al.
Neurotoxicology, 35, 121-128 (2013-01-15)
Chronic exposure to Mn results in the development of a neurological disorder known as manganism characterized by neurological deficits resembling that seen in Parkinsonism. Although dopaminergic neurons within the nigrostriatal pathway appear intact, Mn-induced irregularities in DA transmission have been

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