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Goat Anti-Human IgG γ chain Antibody, HRP conjugate, Species Adsorbed

Chemicon®, from goat

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About This Item

UNSPSC Code:
12352203
eCl@ss:
32160702
NACRES:
NA.46

biological source

goat

Quality Level

conjugate

peroxidase conjugate

antibody form

affinity purified immunoglobulin

antibody product type

secondary antibodies

clone

polyclonal

species reactivity

human

manufacturer/tradename

Chemicon®

technique(s)

ELISA: suitable

shipped in

wet ice

target post-translational modification

unmodified

General description

IgG is one of the most abundant protein in human serum. IgG is important for our defence against microorganisms and the molecules are produced by B-lymphocytes as a part of our adaptive immune response. The IgG molecule has two separate functions; to bind to the pathogen that elicited the response and to recruit other cells and molecules to destroy the antigen. The variability of the IgG pool is generated by somatic recombination.

Specificity

Reacts with with the heavy chain of human IgG. Absorbed for human IgM, IgD and IgA.

Application

Detect Human IgG γ chain using this Goat anti-Human IgG γ chain Antibody, HRP conjugate, Species Adsorbed validated for use in ELISA.

Physical form

Purified by affinity chromatography on pooled human IgG covalently linked to agarose. Liquid in 50% glycerol/50% PBS, pH 7.4. No preservative added.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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Storage Class Code

10 - Combustible liquids

WGK

WGK 2


Certificates of Analysis (COA)

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Lisa M Smith et al.
Journal of virology, 95(23), e0088221-e0088221 (2021-09-23)
Human and simian immunodeficiency virus (HIV and SIV) infections establish lifelong reservoirs of cells harboring an integrated proviral genome. Genome editing CRISPR-associated Cas9 nucleases, combined with SIV-specific guiding RNA (gRNA) molecules, inactivate integrated provirus DNA in vitro and in animal
Adam L Bailey et al.
bioRxiv : the preprint server for biology (2020-11-12)
Epidemiological studies of the COVID-19 pandemic have revealed evidence of cardiac involvement and documented that myocardial injury and myocarditis are predictors of poor outcomes. Nonetheless, little is understood regarding SARS-CoV-2 tropism within the heart and whether cardiac complications result directly

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