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611778

Supelco

Chloroform solution

NMR reference standard, 1% in acetone-d6 (99.9 atom % D), NMR tube size 3 mm × 8 in.

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About This Item

MDL number:
MFCD00000826
UNSPSC Code:
12191502
PubChem Substance ID:
329759379
NACRES:
NA.24

grade

NMR reference standard
analytical standard

Quality Level

200

concentration

1% in acetone-d6 (99.9 atom % D)

technique(s)

NMR: suitable

NMR tube size

3 mm × 8 in.

application(s)

environmental

format

single component solution

SMILES string

ClC(Cl)Cl

InChI

1S/CHCl3/c2-1(3)4/h1H

InChI key

HEDRZPFGACZZDS-UHFFFAOYSA-N

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General description

Chloroform is a colorless liquid, which is volatile in nature. It is generally used as an inhalant anesthetic agent and also as an extraction solvent for oils, fats and greases.
Chloroform is a colorless liquid, which is volatile in nature. It can find applications as an inhalant anesthetic agent. It can also be used as an extraction solvent for oils, fats and greases.

Features and Benefits

1H line shape

Quantity

3 mm O.D. tube contains 0.235 mL.

Signal Word

Danger

Hazard Statements

H225,H319,H336,H351,H373

Hazard Classifications

Carc. 2 - Eye Irrit. 2 - Flam. Liq. 2 - STOT RE 2 Oral - STOT SE 3

Target Organs

Central nervous system, Liver,Kidney

Supplementary Hazards

EUH066

Storage Class Code

3 - Flammable liquids

WGK

WGK 2

Flash Point(F)

1.4 °F - closed cup - Solvent

Flash Point(C)

-17 °C - closed cup - Solvent

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

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Certificates of Analysis (COA)

Lot/Batch Number
MBBC8182
CC0179
EB0243
SZ1312
SZ1805

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National air toxics program the integrated urban strategy, report to Congress (2000)
Causes of death of anesthesiologists from the chloroform era, Volume 1 (1979)
Linde.WH and Mesnick.SP
Causes of death of anesthesiologists from the chloroform era, Volume 1 (1979)
National air toxics program the integrated urban strategy, report to Congress (2000)
Matteo Serino et al.
PloS one, 6(6), e21184-e21184 (2011-06-24)
Daily variations in lipid concentrations in both gut lumen and blood are detected by specific sensors located in the gastrointestinal tract and in specialized central areas. Deregulation of the lipid sensors could be partly involved in the dysfunction of glucose
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