609358
Potassium cyanate-15N
≥98 atom % 15N, ≥95% (CP)
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About This Item
Fórmula lineal:
KOC15N
Número de CAS:
Peso molecular:
82.11
MDL number:
UNSPSC Code:
12352118
PubChem Substance ID:
Productos recomendados
isotopic purity
≥98 atom % 15N
assay
≥95% (CP)
form
solid
mass shift
M+1
SMILES string
[K+].[O-]C#[15N]
InChI
1S/CHNO.K/c2-1-3;/h3H;/q;+1/p-1/i2+1;
InChI key
GKKCIDNWFBPDBW-CGOMOMTCSA-M
Packaging
This product may be available from bulk stock and can be packaged on demand. For information on pricing, availability and packaging, please contact Stable Isotopes Customer Service.
Certificados de análisis (COA)
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F G Schimitel et al.
Neuroscience, 200, 59-73 (2011-11-09)
Dyspnea, hunger for air, and urge to flee are the cardinal symptoms of panic attacks. Patients also show baseline respiratory abnormalities and a higher rate of comorbid and antecedent respiratory diseases. Panic attacks are also precipitated by infusion of sodium
LAIS Allergoid.
G Mistrello
International journal of immunopathology and pharmacology, 18(4 Suppl), 21-24 (2007-12-06)
Małgorzata Iciek et al.
Human & experimental toxicology, 30(10), 1601-1608 (2010-12-24)
Cyanate and its active form isocyanate are formed mainly in the process of nonenzymatic urea biodegradation. Cyanate is capable of protein S- and N-carbamoylation, which can affect their activity. The present studies aimed to demonstrate the effect of cyanate on
Josiane C Cruz et al.
Autonomic neuroscience : basic & clinical, 146(1-2), 47-55 (2009-01-10)
GABAergic, nitrergic and glutamatergic mechanisms in the PVN on the baseline mean arterial pressure (MAP), heart rate (HR) and on the cardiovascular responses to chemoreflex activation in awake rat were evaluated. Chemoreflex was activated with KCN before and after microinjections
H L Liang et al.
Neuroscience, 153(4), 963-974 (2008-04-29)
Parkinson's disease is a common progressive neurodegenerative disorder characterized by the degeneration of dopaminergic neurons in the substantia nigra pars compacta. Mitochondrial dysfunction has been strongly implicated in the pathogenesis of Parkinson's disease. Thus, therapeutic approaches that improve mitochondrial function
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