재조합
expressed in CHO cells
Quality Level
형태
membrane suspension
포장
vial of 50 units
UniProt 수납 번호
배송 상태
dry ice
저장 온도
−70°C
유전자 정보
human ... TACR2(6865)
물리적 형태
Supplied in 50 mM Tris-HCl, pH 7.4, containing 10% glycerol and 1% BSA. Actual concentrations and specific binding capacity are provided with each lot.
분석 메모
Recommended assay conditions are supplied on the product data sheet. This is a bioassayed product.
시험 성적서(COA)
제품의 로트/배치 번호를 입력하여 시험 성적서(COA)을 검색하십시오. 로트 및 배치 번호는 제품 라벨에 있는 ‘로트’ 또는 ‘배치’라는 용어 뒤에서 찾을 수 있습니다.
The Journal of urology, 153(5), 1688-1692 (1995-05-01)
Functional and radioligand binding studies with selective agonists and antagonists were used to investigate tachykinin receptors in the human bladder. Strips of detrusor muscle were contracted by the tachykinins neurokinin A and neuropeptide gamma, and by the NK2 receptor selective
American journal of respiratory cell and molecular biology, 41(6), 651-660 (2009-03-07)
The pathogenesis of sepsis is mediated in part by the pathogen-associated molecular pattern molecule bacterial endotoxin, which stimulates macrophages to sequentially release early (e.g., TNF-alpha, IL-1beta) and late (e.g., high-mobility group box [HMGB] 1 protein) proinflammatory mediators. The recent discovery
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The purpose of the present study was to examine the effects of myricetin on reducing cerebral ischemia injury in a rat model. A rat model of permanent middle cerebral artery occlusion (pMCAO) was used in the present study. Rats were
PloS one, 8(4), e63011-e63011 (2013-05-07)
The peroxisome proliferator-activated receptor subtypes PPARα, PPARβ/δ, PPARγ are members of the steroid hormone receptor superfamily with well-established functions in transcriptional regulation. Here, we describe an unexpected cytoplasmic function of PPARβ/δ. Silencing of PPARβ/δ expression interferes with the expression of
Biochimica et biophysica acta. Molecular cell research, 1868(2), 118917-118917 (2020-12-02)
Duchenne's muscular dystrophy (DMD) is a severe muscle wasting disorder characterized by the loss of dystrophin expression, muscle necrosis, inflammation and fibrosis. Ongoing muscle regeneration is impaired by persistent cytokine stress, further decreasing muscle function. Patients with DMD rarely survive
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