추천 제품
분석
≥95% (HPLC)
mp
192 °C (dec.) (lit.)
solubility
DMSO: ≥20 mg/mL, clear, colorless to faintly yellow
저장 온도
−20°C
SMILES string
Cl[H].COC(=O)C(C)(N)Cc1ccc(O)cc1
InChI
1S/C11H15NO3.ClH/c1-11(12,10(14)15-2)7-8-3-5-9(13)6-4-8;/h3-6,13H,7,12H2,1-2H3;1H
InChI key
OOVDEPZODSXAMU-UHFFFAOYSA-N
유전자 정보
human ... TH(7054)
유사한 제품을 찾으십니까? 방문 제품 비교 안내
애플리케이션
α-Methyl-DL-tyrosine methyl ester hydrochloride has been used to evaluate norepinephrine turnover.
생화학적/생리학적 작용
α-Methyl-DL-tyrosine (α-MT) prevents hyperactivity, sniffing-licking-gnawing syndrome, anorexia and ameliorates certain effects of amphetamine. It inhibits dopamine production and prevents apoptosis stimulated by mutant α-synuclein.
Tyrosine hydroxylase inhibitor.
Storage Class Code
11 - Combustible Solids
WGK
WGK 3
Flash Point (°F)
Not applicable
Flash Point (°C)
Not applicable
개인 보호 장비
Eyeshields, Gloves, type N95 (US)
시험 성적서(COA)
제품의 로트/배치 번호를 입력하여 시험 성적서(COA)을 검색하십시오. 로트 및 배치 번호는 제품 라벨에 있는 ‘로트’ 또는 ‘배치’라는 용어 뒤에서 찾을 수 있습니다.
이미 열람한 고객
Dopamine-dependent neurotoxicity of alpha-synuclein: a mechanism for selective neurodegeneration in Parkinson disease
Nature Medicine, 8(6), 600-600 (2002)
Central sympathetic innervations to visceral and subcutaneous white adipose tissue
American Journal of Physiology. Regulatory, Integrative and Comparative Physiology, 306(6), R375-R386 (2014)
Antiamphetamine effects following inhibition of tyrosine hydroxylase
Journal of Pharmacology and Experimental Therapeutics, 151(3), 339-352 (1966)
Naunyn-Schmiedeberg's archives of pharmacology, 384(1), 39-45 (2011-05-03)
Dopaminergic stabilizers may be conceptualized as drugs with normalizing effects on dopamine-mediated behaviours and neurochemical events. (S)-(-)-OSU6162 (OSU6162) and ACR16 are two structurally related compounds ascribed such properties, principally because of their stabilizing effects on motor activity in rodents. Reports
Neuropharmacology, 56(6-7), 1068-1073 (2009-03-21)
So far, the mechanisms underlying the action of selective serotonin reuptake inhibitors, such as fluoxetine, are not completely understood. Thus, to clarify if fluoxetine has any effect on noradrenergic transmission, we measured the spontaneous firing rate of noradrenergic neurons in
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