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Merck
모든 사진(3)

Key Documents

MAB5206

Sigma-Aldrich

Anti-Amyloid Antibody, β 1-16, clone DE2

culture supernatant, clone DE2, Chemicon®

동의어(들):

Anti-AAA, Anti-ABETA, Anti-ABPP, Anti-AD1, Anti-APPI, Anti-CTFgamma, Anti-CVAP, Anti-PN-II, Anti-PN2, Anti-alpha-sAPP, Anti-preA4

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About This Item

UNSPSC 코드:
12352203
eCl@ss:
32160702
NACRES:
NA.41

생물학적 소스

mouse

Quality Level

항체 형태

culture supernatant

항체 생산 유형

primary antibodies

클론

DE2, monoclonal

종 반응성

monkey, bovine, human

제조업체/상표

Chemicon®

기술

ELISA: suitable
immunohistochemistry: suitable (paraffin)
western blot: suitable

동형

IgG

UniProt 수납 번호

배송 상태

dry ice

타겟 번역 후 변형

unmodified

유전자 정보

human ... APP(351)

애플리케이션

Anti-Amyloid Antibody, β 1-16, clone DE2 is an antibody against Amyloid for use in ELISA, WB, IH(P).
Western blot: 1:10-1:300 Immunohistochemistry: 1:10-1:200 on formaldehyde fixed, paraffin embedded tissue, requires formic acid or microwave pretreatment to stain Alzheimer′s brain senile plaques. ELISA Immunoprecipitation

Optimal working dilutions must be determined by end user.

물리적 형태

Culture supernatant. Liquid containing 0.02% sodium azide.

법적 정보

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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Storage Class Code

10 - Combustible liquids

WGK

WGK 1


시험 성적서(COA)

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문서 라이브러리 방문

Alexander J Ehrenberg et al.
Acta neuropathologica, 141(5), 651-666 (2021-03-08)
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S100A9 knockout decreases the memory impairment and neuropathology in crossbreed mice of Tg2576 and S100A9 knockout mice model.
Kim, HJ; Chang, KA; Ha, TY; Kim, J; Ha, S; Shin, KY; Moon, C; Nacken, W; Kim, HS; Suh, YH
Testing null
Sandra Grau et al.
Proceedings of the National Academy of Sciences of the United States of America, 102(17), 6021-6026 (2005-04-28)
The defining features of the widely conserved HtrA (high temperature requirement) family of serine proteases are the combination of a catalytic protease domain with one or more C-terminal PDZ domains and reversible zymogen activation. Even though HtrAs have previously been
Hazal Haytural et al.
Brain communications, 3(2), fcab079-fcab079 (2021-05-21)
Synaptic degeneration has been reported as one of the best pathological correlates of cognitive deficits in Alzheimer's disease. However, the location of these synaptic alterations within hippocampal sub-regions, the vulnerability of the presynaptic versus postsynaptic compartments, and the biological mechanisms

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