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Merck
모든 사진(4)

주요 문서

07-674

Sigma-Aldrich

Anti-Na+K+ ATPase α-2 Antibody

serum, Upstate®

동의어(들):

Anti-Anti-DEE98, Anti-Anti-FARIMPD, Anti-Anti-FHM2, Anti-Anti-MHP2

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About This Item

UNSPSC 코드:
12352203
eCl@ss:
32160702
NACRES:
NA.41

생물학적 소스

rabbit

Quality Level

항체 형태

serum

항체 생산 유형

primary antibodies

클론

polyclonal

종 반응성

rat

제조업체/상표

Upstate®

기술

western blot: suitable

동형

IgG

NCBI 수납 번호

UniProt 수납 번호

배송 상태

dry ice

타겟 번역 후 변형

unmodified

유전자 정보

human ... ATP1A2(477)

특이성

Na+/K+ ATPase α-2
Predicted to crossreact with human, mouse, dog and monkey based on sequence homology.

면역원

KLH-conjugated, synthetic peptide (CKAGQENISVSKRDT) corresponding to amino acids 432-445 of human Na+K+ ATPase α-2, with an N-terminal cysteine added for conjugation purposes.

애플리케이션

Anti-Na+K+ ATPase α-2 Antibody is an antibody against Na+K+ ATPase α-2 for use in WB.
Research Category
Neuroscience
Research Sub Category
Ion Channels & Transporters

품질

Routinely evaluated by immunoblot with rat brain microsomal preparation (catalog# 12-144)

표적 설명

Mr 105kDa

물리적 형태

Antiserum

저장 및 안정성

Stable for 2 years at -20°C from date of shipment

법적 정보

UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany

면책조항

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

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Storage Class Code

12 - Non Combustible Liquids

WGK

WGK 1

Flash Point (°F)

Not applicable

Flash Point (°C)

Not applicable


시험 성적서(COA)

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문서 라이브러리에서 최근에 구매한 제품에 대한 문서를 찾아보세요.

문서 라이브러리 방문

Mads S Toustrup-Jensen et al.
The Journal of biological chemistry, 289(6), 3186-3197 (2013-12-21)
The neurological disorders familial hemiplegic migraine type 2 (FHM2), alternating hemiplegia of childhood (AHC), and rapid-onset dystonia parkinsonism (RDP) are caused by mutations of Na(+),K(+)-ATPase α2 and α3 isoforms, expressed in glial and neuronal cells, respectively. Although these disorders are
A Mobasheri et al.
Bioscience reports, 20(2), 51-91 (2000-08-31)
Na+, K+-ATPase is ubiquitously expressed in the plasma membrane of all animal cells where it serves as the principal regulator of intracellular ion homeostasis. Na+, K+-ATPase is responsible for generating and maintaining transmembrane ionic gradients that are of vital importance
Modulation of endothelial cell KCa3.1 channels during endothelium-derived hyperpolarizing factor signaling in mesenteric resistance arteries.
Dora, KA; Gallagher, NT; McNeish, A; Garland, CJ
Circulation Research null
The molecular genetics of migraine.
Wessman, Maija, et al.
Annals of Medicine, 36, 462-473 (2004)
Post-ischemic stimulation of 2-deoxyglucose uptake in rat myocardium: role of translocation of Glut-4
Montessuit, C, et al
Journal of Molecular and Cellular Cardiology, 30, 393-403 (1998)

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