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Key Documents

Safety Information

N0271

Millipore

Filter Membrane

Nitrocellulose, Thin-film membrane, pore size 0.45 μm, diam. 47 mm

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About This Item

UNSPSC Code:
41104900
PubChem Substance ID:
24897419
NACRES:
NB.24

Product Name

Filter membranes, nitrocellulose, TF membrane, pore size 0.45 μm, diam. 47 mm

material

TF membrane
nitrocellulose membrane
plain white filter

manufacturer/tradename

Millipore HATF04700

diam.

47 mm

pore size

0.45 μm

detection method

chemiluminescent
colorimetric
fluorometric
radioactive

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General description

Biologically inert nitrocellulose membranes containing a small amount of cellulose acetate for improved handling. Autoclavable to 121°C; use below 75°C. Chemically compatible with dilute acids and bases, hydrocarbons, and non-polar liquids. MF-Millipore membranes have water extractables <5%, including a Triton wetting agent TF-Millipore membranes are Triton-free and have even lower water extractables than MF membranes. See Immobilon-NC in the Electrophoresis Equipment section for nitrocellulose blotting membranes.

Pictograms

Flame
GHS02

Signal Word

Danger

Hazard Statements

H228

Precautionary Statements

P210

Hazard Classifications

Flam. Sol. 1

Storage Class Code

4.1B - Flammable solid hazardous materials

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Regulatory Listings

Regulatory Listings are mainly provided for chemical products. Only limited information can be provided here for non-chemical products. No entry means none of the components are listed. It is the user’s obligation to ensure the safe and legal use of the product.

FSL

Group 5: Self-reactive substances
Nitro compounds
Hazardous rank I
1st self-reactive materials

JAN Code

N0271-100EA:
N0271-100EAPW:
N0271-BULK:

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Certificates of Analysis (COA)

Lot/Batch Number

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Dinesh Chandra et al.
Clinical and experimental immunology, 154(2), 224-234 (2008-09-10)
The failure of Leishmania, an intracellular pathogen, to stimulate a pro-inflammatory response following entry into macrophages has been well reported. This occurs in spite of the fact that ligands for the toll-like receptors (TLR) have been recently shown on the

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