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C1900

Sigma-Aldrich

Protoporphyrin IX cobalt chloride

Synonym(s):

Cobaltic protoporphyrin IX chloride

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About This Item

Linear Formula:
C34H32CoN4O4Cl
CAS Number:
102601-60-5
Molecular Weight:
655.03
MDL number:
MFCD00216774
UNSPSC Code:
12352202
PubChem Substance ID:
329774912
NACRES:
NA.22

form

powder

reaction suitability

core: cobalt
reagent type: catalyst

storage temp.

2-8°C

SMILES string

Cc1c(CCC(O)=O)c2cc3c(CCC(O)=O)c(C)c4cc5nc(cc6c(C=C)c(C)c(cc1n2)n6[Co](Cl)n34)c(C)c5C=C

InChI

1S/C34H34N4O4.ClH.Co/c1-7-21-17(3)25-13-26-19(5)23(9-11-33(39)40)31(37-26)16-32-24(10-12-34(41)42)20(6)28(38-32)15-30-22(8-2)18(4)27(36-30)14-29(21)35-25;;/h7-8,13-16H,1-2,9-12H2,3-6H3,(H4,35,36,37,38,39,40,41,42);1H;/q;;+3/p-3/b25-13-,26-13-,27-14-,28-15-,29-14-,30-15-,31-16-,32-16-;;

InChI key

WWNXKQFLDNSNDN-HXFTUNQESA-K

Application

Reactant involved in studies of:
  • Enzyme models of hCBS heme group function
  • Electrocatalytic reduction of oxygen at electropolymerized films of porphyrins
  • Electrochemical redox behavior
  • Nitrogen(II) oxide coordination by metal porphyrins

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Regulatory Listings

Regulatory Listings are mainly provided for chemical products. Only limited information can be provided here for non-chemical products. No entry means none of the components are listed. It is the user’s obligation to ensure the safe and legal use of the product.

PRTR

Class I Designated Chemical Substances

ISHL Indicated Name

Substances Subject to be Indicated Names

ISHL Notified Names

Substances Subject to be Notified Names

JAN Code

C1900-100MG:4548174003428
C1900-25MG:
C1900-BULK:
C1900-VAR:
C1900-250MG:

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Certificates of Analysis (COA)

Lot/Batch Number
MKCQ2382
MKCP0241
MKCM7302
MKCL3056
MKCG6419

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Shuang Li et al.
Antioxidants & redox signaling, 30(13), 1635-1650 (2018-08-08)
The risk factors promoting acute kidney injury (AKI) to chronic kidney disease (CKD) progression remain largely unknown. The aim of the present study was to investigate whether hyperhomocysteinemia (Hhcy) accelerates the development of renal fibrosis after AKI. Hhcy aggravated ischemia-reperfusion-induced

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