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Key Documents

HT110232

Sigma-Aldrich

Eosin Y Solution, Aqueous

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About This Item

Codice UNSPSC:
41116124
NACRES:
NA.47

Forma fisica

solution

Livello qualitativo

Durata

Expiry date on the label.

IVD

for in vitro diagnostic use

Concentrazione

0.5 % (w/v) in water

applicazioni

hematology
histology

Temperatura di conservazione

room temp

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Applicazioni

General purpose cytoplasmic counterstain. Used with hematoxylin and eosin staining.

Altre note

Certified Eosin Y, 0.5% (w/v) in water. Not acidified.

Codice della classe di stoccaggio

10 - Combustible liquids

Classe di pericolosità dell'acqua (WGK)

WGK 2

Punto d’infiammabilità (°F)

Not applicable

Punto d’infiammabilità (°C)

Not applicable

Dispositivi di protezione individuale

Eyeshields, Gloves, multi-purpose combination respirator cartridge (US)


Certificati d'analisi (COA)

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Oliver Hachmöller et al.
Journal of trace elements in medicine and biology : organ of the Society for Minerals and Trace Elements (GMS), 44, 71-75 (2017-10-03)
The influence of rhodanine and haematoxylin and eosin (HE) staining on the copper distribution and concentration in liver needle biopsy samples originating from patients with Wilson's disease (WD), a rare autosomal recessive inherited disorder of the copper metabolism, is investigated.
Wei-Ting Chen et al.
Cell, 182(4), 976-991 (2020-07-24)
Although complex inflammatory-like alterations are observed around the amyloid plaques of Alzheimer's disease (AD), little is known about the molecular changes and cellular interactions that characterize this response. We investigate here, in an AD mouse model, the transcriptional changes occurring
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Skeletal muscle tissue has an enormous regenerative capacity that is instrumental for a successful defense against muscle injury and wasting. The peroxisome proliferator-activated receptor γ coactivator 1α (PGC-1α) exerts therapeutic effects in several muscle pathologies, but its role in damage-induced
Marshall W Hogarth et al.
Nature communications, 8, 14143-14143 (2017-02-01)
Duchenne muscular dystrophy (DMD) is characterized by muscle degeneration and progressive weakness. There is considerable inter-patient variability in disease onset and progression, which can confound the results of clinical trials. Here we show that a common null polymorphism (R577X) in
Federica Francescangeli et al.
Journal of experimental & clinical cancer research : CR, 39(1), 2-2 (2020-01-09)
Quiescent/slow cycling cells have been identified in several tumors and correlated with therapy resistance. However, the features of chemoresistant populations and the molecular factors linking quiescence to chemoresistance are largely unknown. A population of chemoresistant quiescent/slow cycling cells was isolated

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