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SAB4300039

Sigma-Aldrich

Anti-phospho-STAT6 (pThr645) antibody produced in rabbit

affinity isolated antibody

Synonym(s):

Anti-D12S1644 antibody produced in rabbit, Anti-IL-4-STAT antibody produced in rabbit, Anti-STAT6B antibody produced in rabbit, Anti-STAT6C antibody produced in rabbit, Anti-signal transducer and activator of transcription 6, interleukin-4 induced antibody produced in rabbit

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About This Item

MDL number:
UNSPSC Code:
12352203
NACRES:
NA.41

biological source

rabbit

Quality Level

conjugate

unconjugated

antibody form

affinity isolated antibody

antibody product type

primary antibodies

clone

polyclonal

form

buffered aqueous solution

mol wt

~110 kDa

species reactivity

human

concentration

1 mg/mL

technique(s)

immunohistochemistry (formalin-fixed, paraffin-embedded sections): 1:50-1:100

isotype

IgG

immunogen sequence

(P-A-TP-I-K)

NCBI accession no.

UniProt accession no.

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

phosphorylation (pThr645)

Gene Information

human ... STAT6(6778)

Related Categories

Immunogen

Peptide sequence around phosphorylation site of threonine 645 (P-A-T(p)-I-K), according to the protein STAT6.

Features and Benefits

Evaluate our antibodies with complete peace of mind. If the antibody does not perform in your application, we will issue a full credit or replacement antibody. Learn more.

Target description

Carries out a dual function: signal transduction and activation of transcription. Involved in interleukin-4 signalling.

Physical form

Solution in phosphate-buffered saline containing 0.02% sodium azide and 50% glycerol

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Storage Class Code

10 - Combustible liquids

WGK

WGK 1

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable


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E Yaneth Osorio et al.
PLoS pathogens, 10(6), e1004165-e1004165 (2014-06-27)
Host arginase 1 (arg1) expression is a significant contributor to the pathogenesis of progressive visceral leishmaniasis (VL), a neglected tropical disease caused by the intracellular protozoan Leishmania donovani. Previously we found that parasite-induced arg1 expression in macrophages was dependent on
Sun Hee Jin et al.
Toxicology and applied pharmacology, 280(2), 199-206 (2014-08-30)
Keratinocytes are the major cellular components of human epidermis and play a key role in the modulating cutaneous inflammation and toxic responses. In human chronic skin diseases, the common skin inflammatory phenotypes like skin barrier disruption and epidermal hyperplasia are

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