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EMU005241

Sigma-Aldrich

MISSION® esiRNA

targeting mouse Prkdc

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About This Item

UNSPSC Code:
41105324
NACRES:
NA.51

description

Powered by Eupheria Biotech

Quality Level

100

product line

MISSION®

form

lyophilized powder

esiRNA cDNA target sequence

ACCACTGGCCATTTTCAGAGACGGGAGCATCAAGATTCCATGACCCAGGATGACATCATGGAGTTAGAGATGGATGAGCTCAATCAACATGAATGTATGGCTCCCATGATAGCCCTGATTAAGCACATGCAGAGAAATGTGATTGCACCTAAGGGAGAAGAGGGTTCAATACCAAAAGATCTTCCACCGTGGATGAAATTTCTTCATGACAAACTAGGAAATGCATCAGTATCTTTAAATATCTGTCTCTTCTTAGCCAAGCTTGTTATTAATACAGAAGAGGTCTTTCGCCCTTATGCAAAGCACTGGCTCAGCCCCCTGCTACAGCTAGCTGTTTGCGAGAACAACAGAGAAGGAATTCACTACATGATGGTAGAAATAGTAGCTACCATTCTCTCTTGGACTGGC

Ensembl | mouse accession no.

ENSMUSG00000022672

NCBI accession no.

NM_011159

shipped in

ambient

storage temp.

−20°C

Gene Information

mouse ... PRKDC(19090) , Prkdc(19090)

Related Categories

General description

MISSION® esiRNA are endoribonuclease prepared siRNA. They are a heterogeneous mixture of siRNA that all target the same mRNA sequence. These multiple silencing triggers lead to highly-specific and effective gene silencing.

For additional details as well as to view all available esiRNA options, please visit SigmaAldrich.com/esiRNA.

Legal Information

MISSION is a registered trademark of Merck KGaA, Darmstadt, Germany

Storage Class Code

10 - Combustible liquids

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Certificates of Analysis (COA)

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Ewa Kotula et al.
Cell cycle (Georgetown, Tex.), 14(12), 1961-1972 (2015-05-29)
The DNA-dependent protein kinase catalytic subunit (DNA-PKcs) plays a major role in DNA damage signaling and repair and is also frequently overexpressed in tumor metastasis. We used isogenic cell lines expressing different levels of DNA-PKcs to investigate the role of
Christina Hausmann et al.
Oncotarget, 6(33), 34592-34605 (2015-10-16)
The prognosis is generally poor for patients suffering from glioblastoma multiforme (GBM) due to radiation and drug resistance. Prosurvival signaling originating from focal adhesion hubs essentially contributes to therapy resistance and tumor aggressiveness. As the underlying molecular mechanisms remain largely

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