05091
3,5-Dimethylpyrazole
produced by Wacker Chemie AG, Burghausen, Germany, ≥99.0% (GC)
Synonym(s):
3,5-Dimethyl-1H-pyrazole
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About This Item
Recommended Products
grade
produced by Wacker Chemie AG, Burghausen, Germany
Quality Level
Assay
≥99.0% (GC)
bp
218 °C (lit.)
mp
105-108 °C (lit.)
SMILES string
Cc1cc(C)[nH]n1
InChI
1S/C5H8N2/c1-4-3-5(2)7-6-4/h3H,1-2H3,(H,6,7)
InChI key
SDXAWLJRERMRKF-UHFFFAOYSA-N
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Related Categories
Signal Word
Warning
Hazard Statements
Precautionary Statements
Hazard Classifications
Acute Tox. 4 Oral - STOT RE 2
Target Organs
Liver
Storage Class Code
13 - Non Combustible Solids
WGK
WGK 3
Personal Protective Equipment
dust mask type N95 (US), Eyeshields, Gloves
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Autophagy, 3(1), 26-27 (2006-09-12)
Autophagy is a major intracellular degradation/recycling system ubiquitous in eukaryotic cells. It contributes to the turnover of cellular components by delivering portions of the cytoplasm and organelles to lysosomes, where they are digested. Starvation-induced autophagy is required for maintaining an
Journal of endocrinological investigation, 7(4), 277-281 (1984-08-01)
The inhibitory and stimulatory effects of somatostatin analogues on growth hormone secretion have been studied in the sheep. Plasma immunoreactive growth hormone (GH) was stimulated by the iv administration of the antilipolytic compound 3,5-dimethylpyrazole and was followed by infusion of
Effects of antilipolytic agents on peroxisomal beta-oxidation of fatty acids in rat liver.
Biochemical pharmacology, 32(11), 1807-1809 (1983-06-01)
The regulation of liver protein degradation by aminoacids in vivo. Effects of glutamine and leucine.
Archives of physiology and biochemistry, 103(4), 512-515 (1995-08-01)
The effects in vivo of the two major in vitro regulatory aminoacids, leucine and glutamine, on liver protein degradation were explored in male young adult Sprague Dawley rats. Protein degradation was stimulated by the injection of the antilipolytic drug 3,5
Metabolism: clinical and experimental, 51(1), 110-114 (2002-01-10)
This study intended to test the hypothesis that intracellular lipolysis in the pancreatic beta cells is implicated in the regulation of insulin secretion stimulated by nutrient secretagogues or cyclic adenosine monophosphate (cAMP) agonists. Indeed, although lipid signaling molecules were repeatedly
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