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657012

Sigma-Aldrich

Anti-Tyrosine Hydroxylase Rabbit pAb

liquid, Calbiochem®

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About This Item

Code UNSPSC :
12352203

Source biologique

rabbit

Niveau de qualité

Forme d'anticorps

purified antibody

Type de produit anticorps

primary antibodies

Clone

polyclonal

Forme

liquid

Ne contient pas

preservative

Réactivité de l'espèce (prédite par homologie)

all

Fabricant/nom de marque

Calbiochem®

Conditions de stockage

OK to freeze
avoid repeated freeze/thaw cycles

Isotype

IgG

Conditions d'expédition

wet ice

Température de stockage

−70°C

Modification post-traductionnelle de la cible

unmodified

Description générale

Affinity purified rabbit polyclonal antibody. Recognizes the ~60 kDa tyrosine hydroxylase protein.
Recognizes the ~60 kDa tyrosine hydroxylase protein in okadaic acid-treated PC12 cells.
This Anti-Tyrosine Hydroxylase Rabbit pAb is validated for use in Dot Blot, Immunoblotting, Immunofluorescence, Frozen Sections for the detection of Tyrosine Hydroxylase.

Immunogène

Rat
purified, SDS-denatured rat pheochromocytoma tyrosine hydroxylase

Application

Dot Blot (1:1000)

Immunoblotting (1:1000)

Immunofluorescence (1:1000)

Frozen Sections (1:1000; see application references)

Avertissement

Toxicity: Standard Handling (A)

Forme physique

In 150 mM NaCl, 10 mM HEPES, 50% glycerol, 0.01% BSA, pH 7.5.

Reconstitution

Following initial thaw, aliquot and freeze (-70°C).

Informations légales

CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany

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Code de la classe de stockage

10 - Combustible liquids

Classe de danger pour l'eau (WGK)

WGK 1


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The in vivo firing patterns of ventral midbrain dopamine neurons are controlled by afferent and intrinsic activity to generate sensory cue and prediction error signals that are essential for reward-based learning. Given the absence of in vivo intracellular recordings during
Ximena I Salinas-Hernández et al.
eLife, 7 (2018-11-14)
Extinction of fear responses is critical for adaptive behavior and deficits in this form of safety learning are hallmark of anxiety disorders. However, the neuronal mechanisms that initiate extinction learning are largely unknown. Here we show, using single-unit electrophysiology and
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The Journal of comparative neurology, 526(3), 425-438 (2017-10-22)
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Saurabh Khasnavis et al.
The Journal of biological chemistry, 288(29), 20843-20855 (2013-06-08)
Although Parkinson disease (PD) is a progressive neurodegenerative disorder, available animal models do not exhibit irreversible neurodegeneration, and this is a major obstacle in finding out an effective drug against this disease. Here we delineate a new irreversible model to

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