MAB10216
Anti-Flavivirus Group Antigen Antibody, clone D1-4G2-4-15
ascites fluid, clone D1-4G2-4-15, Chemicon®
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About This Item
Código UNSPSC:
12352203
eCl@ss:
32160702
Productos recomendados
origen biológico
mouse
forma del anticuerpo
ascites fluid
tipo de anticuerpo
primary antibodies
clon
D1-4G2-4-15, monoclonal
reactividad de especies
human
fabricante / nombre comercial
Chemicon®
técnicas
immunofluorescence: suitable
isotipo
IgG1
IgG2a
Condiciones de envío
dry ice
Descripción general
The viruses of the family Flaviviridae are important arthropod-borne viruses in both human and veterinary medicine. They are transmitted by mosquito and ticks and usually are maintained in a transmission cycle in nature. They produce a broad spectrum of clinical responses in humans ranging from asymptomatic infection to fulminant encephalitis or haemorrhagic fever. Nearly 60 flaviviruses are known to exist but many are yet to be shown to cause disease in humans.
Especificidad
Recognises flavivirus group specific antigens (Dengue virus, West Nile Virus, Japanese Encephalitis, Yellow Fever Virus, Zika virus etc). It binds to the fusion loop at the extremity of domain II of protein E.
Inmunógeno
Dengue virus type 2 antigens
Aplicación
Anti-Flavivirus Group Antigen Antibody, clone D1-4G2-4-15 detects level of Flavivirus Group Antigen & has been published & validated for use in IF.
Información legal
CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany
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Código de clase de almacenamiento
10 - Combustible liquids
Clase de riesgo para el agua (WGK)
WGK 1
Punto de inflamabilidad (°F)
Not applicable
Punto de inflamabilidad (°C)
Not applicable
Certificados de análisis (COA)
Busque Certificados de análisis (COA) introduciendo el número de lote del producto. Los números de lote se encuentran en la etiqueta del producto después de las palabras «Lot» o «Batch»
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Qian Chen et al.
Nature communications, 11(1), 2967-2967 (2020-06-13)
The recent outbreak of Zika virus (ZIKV) was associated with birth defects and pregnancy loss when maternal infection occurs in early pregnancy, but specific mechanisms driving placental insufficiency and subsequent ZIKV-mediated pathogenesis remain unclear. Here we show, using large scale
V Haridas et al.
PLoS neglected tropical diseases, 7(1), e2005-e2005 (2013-01-26)
Japanese encephalitis virus (JEV) is a major cause of viral encephalitis in South and South-East Asia. Lack of antivirals and non-availability of affordable vaccines in these endemic areas are a major setback in combating JEV and other closely related viruses
A Viral Polymerase Inhibitor Reduces Zika Virus Replication in the Reproductive Organs of Male Mice.
Sofie Jacobs et al.
International journal of molecular sciences, 20(9) (2019-04-30)
In humans, Zika virus and viral RNA have been detected in semen up to 2.2 months and 6 months post infection (pi), respectively. Although the contribution of sexual transmission to the spread of ZIKV is too low to sustain an
Andreas S Puschnik et al.
Cell reports, 21(11), 3032-3039 (2017-12-16)
The mosquito-borne flaviviruses include important human pathogens such as dengue, Zika, West Nile, and yellow fever viruses, which pose a serious threat for global health. Recent genetic screens identified endoplasmic reticulum (ER)-membrane multiprotein complexes, including the oligosaccharyltransferase (OST) complex, as
José A Aguilar-Briseño et al.
Nature communications, 11(1), 3177-3177 (2020-06-25)
Vascular permeability and plasma leakage are immune-pathologies of severe dengue virus (DENV) infection, but the mechanisms underlying the exacerbated inflammation during DENV pathogenesis are unclear. Here, we demonstrate that TLR2, together with its co-receptors CD14 and TLR6, is an innate
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