05-479
Anti-Cytochrome C Antibody, clone C-7
ascites fluid, clone C-7, Upstate®
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About This Item
UNSPSC-Code:
12352203
eCl@ss:
32160702
Empfohlene Produkte
Biologische Quelle
mouse
Antikörperform
ascites fluid
Antikörper-Produkttyp
primary antibodies
Klon
C-7, monoclonal
Speziesreaktivität
human, horse
Hersteller/Markenname
Upstate®
Methode(n)
immunohistochemistry: suitable
western blot: suitable
Isotyp
IgG
NCBI-Hinterlegungsnummer
UniProt-Hinterlegungsnummer
Versandbedingung
dry ice
Posttranslationale Modifikation Target
unmodified
Angaben zum Gen
human ... CYCS(54205)
Spezifität
Cytochrome c
Immunogen
Horse cytochrome c conjugated to KLH
Anwendung
Research Category
Apoptose & Krebs
Stoffwechsel
Apoptose & Krebs
Stoffwechsel
Research Sub Category
Apoptose - Weiterführende Produkte
Enzyme & Biochemie
Apoptose - Weiterführende Produkte
Enzyme & Biochemie
Detect Cytochrome C using this Anti-Cytochrome C Antibody, clone C-7 validated for use in WB, IH.
Qualität
routinely evaluated by immunoblot on RIPA lysates from A431 cells
Zielbeschreibung
14kDa
Verlinkung
Replaces: 04-1043
Physikalische Form
Ascites
mouse ascites containing 0.05% sodium azide
Lagerung und Haltbarkeit
2 years at -20°C
Rechtliche Hinweise
UPSTATE is a registered trademark of Merck KGaA, Darmstadt, Germany
Haftungsausschluss
Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.
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Produkt-Nr.
Beschreibung
Preisangaben
Lagerklassenschlüssel
12 - Non Combustible Liquids
WGK
WGK 1
Flammpunkt (°C)
Not applicable
Analysenzertifikate (COA)
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Structural basis for the binding of an anti-cytochrome c antibody to its antigen: crystal structures of FabE8-cytochrome c complex to 1.8 A resolution and FabE8 to 2.26 A resolution.
Mylvaganam, S E, et al.
Journal of Molecular Biology, 281, 301-322 (1998)
J E Springer et al.
Nature medicine, 5(8), 943-946 (1999-07-30)
Traumatic spinal cord injury often results in complete loss of voluntary motor and sensory function below the site of injury. The long-term neurological deficits after spinal cord trauma may be due in part to widespread apoptosis of neurons and oligodendroglia
Rui Liu et al.
Molecular cancer therapeutics, 14(9), 2090-2102 (2015-07-05)
Tumor adaptive resistance to therapeutic radiation remains a barrier for further improvement of local cancer control. SIRT3, a member of the sirtuin family of NAD(+)-dependent protein deacetylases in mitochondria, promotes metabolic homeostasis through regulation of mitochondrial protein deacetylation and plays
So-Yeon Kim et al.
Oncotarget, 8(39), 64964-64973 (2016-08-05)
Small molecules to selectively induce cell death of undifferentiated human pluripotent stem cells (hPSCs) have been developed with the aim of lowering the risk of teratoma formation during hPSC-based cell therapy. In this context, we have reported that Quercetin (QC)
Xiao-Qing Ding et al.
Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology, 41(2), 835-848 (2017-02-20)
The present study investigated whether the transient receptor potential melastatin 4 (TRPM4) channel plays a role in high salt diet (HSD)-induced endothelial injuries. Western blotting and immunofluorescence were used to examine TRPM4 expression in the mesenteric endothelium of Dahl salt-sensitive
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