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  • Combined replacement effects of human modified β-hexosaminidase B and GM2 activator protein on GM2 gangliosidoses fibroblasts.

Combined replacement effects of human modified β-hexosaminidase B and GM2 activator protein on GM2 gangliosidoses fibroblasts.

Biochemistry and biophysics reports (2016-06-08)
Keisuke Kitakaze, Chikako Tasaki, Youichi Tajima, Takatsugu Hirokawa, Daisuke Tsuji, Hitoshi Sakuraba, Kohji Itoh
ABSTRACT

GM2 gangliosidoses are autosomal recessive lysosomal storage diseases (LSDs) caused by mutations in the

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Sigma-Aldrich
Anti-actinaβ monoclonale, clone AC-74, ascites fluid
Sigma-Aldrich
Anti-GM2A antibody produced in rabbit, Prestige Antibodies® Powered by Atlas Antibodies, affinity isolated antibody, buffered aqueous glycerol solution