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Merck
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Principaux documents

4060

Millipore

Inosine

A metabolite of adenosine that acts as a potent coronary vasodilator. Exhibits a positive inotropic action on heart muscle and can antagonize the ouabain-induced arrhythmias.

Synonyme(s) :

Inosine, Hypoxanthine Riboside

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About This Item

Formule empirique (notation de Hill):
C10H12N4O5
Numéro CAS:
58-63-9
Poids moléculaire :
268.23
Numéro MDL:
MFCD00066770
Code UNSPSC :
12352208
Nomenclature NACRES :
NA.51

Niveau de qualité

100

Essai

≥99% (by assay)

Forme

solid

Fabricant/nom de marque

Calbiochem®

Conditions de stockage

OK to freeze

Couleur

white

Solubilité

water: soluble

Conditions d'expédition

ambient

Température de stockage

15-25°C

InChI

1S/C10H12N4O5/c15-1-4-6(16)7(17)10(19-4)14-3-13-5-8(14)11-2-12-9(5)18/h2-4,6-7,10,15-17H,1H2,(H,11,12,18)

Clé InChI

UGQMRVRMYYASKQ-UHFFFAOYSA-N

Description générale

A metabolite of adenosine that acts as a potent coronary vasodilator. Exhibits a positive inotropic action on heart muscle and can antagonize the ouabain-induced arrhythmias.
Correlates well with myocardial contractile performance.

Avertissement

Toxicity: Standard Handling (A)

Autres remarques

Aviado, D.M. 1983. J. Pharmacol. 14 (suppl. 3), 47.

Informations légales

CALBIOCHEM is a registered trademark of Merck KGaA, Darmstadt, Germany

Code de la classe de stockage

11 - Combustible Solids

Classe de danger pour l'eau (WGK)

WGK 2

Point d'éclair (°F)

Not applicable

Point d'éclair (°C)

Not applicable

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Juan M Roldan-Romero et al.
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The mammalian target of rapamycin (mTOR) pathway inhibitors are key drugs for the treatment of many tumor types, however, there are no predictive biomarkers in clinical use. Here, we performed a molecular and immunohistochemical characterization of key mTOR pathway components
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WW domain-binding protein 2 (WBP2) has been demonstrated as oncogenic in breast cancer. Many studies have revealed the WBP2 gene as a high-risk gene for leukoariaosis and cerebral white matter lesions is important in the pathologic stage of glioma development.
Dominick G A Burton et al.
PloS one, 8(6), e68003-e68003 (2013-07-11)
Androgen deprivation (AD) is an effective method for initially suppressing prostate cancer (PC) progression. However, androgen-refractory PC cells inevitably emerge from the androgen-responsive tumor, leading to incurable disease. Recent studies have shown AD induces cellular senescence, a phenomenon that is
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